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Blood coagulation in glomerulonephritis
International Urology and Nephrology, 1986Different parameters of coagulation were studied in 71 patients with glomerulonephritis. In comparison with normal subjects they had lower platelet counts, lower adhesion index and decreased aggregation, decreased partial thromboplastin time (46.4 per cent of patients), increased reptilase time (47.8 per cent) and other disorders. Plasma fibrin monomer
N, Belovezhdov, R, Robeva, V, Genova
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The kinetics of blood coagulation
The Bulletin of Mathematical Biophysics, 1948The kinetics of the blood coagulation system have been formulated and an expression obtained for the “prothrombin time” in terms of the concentrations of the components of the system. A linear plot of data obtained from plasma dilution curves gives the values of the parameters of the system, and yields a mathematical method of comparing relative ...
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The Coagulation of Blood and Hemostasis
Annual Review of Physiology, 1950Since the number of papers on coagulation has greatly in creased, the reviewer, in order to avoid merely cataloging them with telegraphic brevity, was compelled to select mainly those which contribute specifically to the physiology of blood clotting.
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Regulation of blood coagulation
Biochimica et Biophysica Acta (BBA) - Protein Structure and Molecular Enzymology, 2000The protein C anticoagulant pathway converts the coagulation signal generated by thrombin into an anticoagulant response through the activation of protein C by the thrombin-thrombomodulin (TM) complex. The activated protein C (APC) thus formed interacts with protein S to inactivate two critical coagulation cofactors, factors Va and VIIIa, thereby ...
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Transfusion and Apheresis Science, 2018
Hemostasis is the physiological control of bleeding and is initiated by subendothelial exposure. Platelets form the primary vascular seal in three stages (localization, stimulation and aggregation), which are triggered by specific interactions between platelet surface receptors and constituents of the subendothelial matrix.
Edward L G, Pryzdial +5 more
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Hemostasis is the physiological control of bleeding and is initiated by subendothelial exposure. Platelets form the primary vascular seal in three stages (localization, stimulation and aggregation), which are triggered by specific interactions between platelet surface receptors and constituents of the subendothelial matrix.
Edward L G, Pryzdial +5 more
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American Journal of Obstetrics and Gynecology, 1987
Blood coagulation consists of several interrelated reactions involving platelets and blood vessels, the classical cascade, and the fibrinolytic system. There is a complex of inhibiting influences on each of these schemes, and there are interrelationships among these inhibitory influences.
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Blood coagulation consists of several interrelated reactions involving platelets and blood vessels, the classical cascade, and the fibrinolytic system. There is a complex of inhibiting influences on each of these schemes, and there are interrelationships among these inhibitory influences.
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Erythrocytes and Blood Coagulation
Thrombosis and Haemostasis, 1974SummaryThe action of intact and hemolyzed red cells in in vitro clotting systems is analyzed against the background of the clinical manifestations of red cell procoagulants in incompatible transfusions and hemolytic anemias in man and in experimental animals.
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The Genetics of Blood Coagulation
1983Hemostasis is the term applied to the process that regulates the loss of blood from the circulatory system following injury. It involves three interrelated physiological mechanisms: constriction of blood vessels, aggregation of blood platelets to damaged subendothelial surfaces, and the formation of fibrin clots.
J B, Graham +3 more
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Hemodialysis International, 2001
The endothelium is the principal anti‐thrombotic mechanism, providing a non wettable surface and generating potent vasodilators (nitric oxide and prostacyclin) and clotting inhibitors [thrombomodulin and tissue factor pathway inhibitor (TFPI)]. When the integrity of the endothelium is breached, vasoconstriction occurs through neural and chemical ...
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The endothelium is the principal anti‐thrombotic mechanism, providing a non wettable surface and generating potent vasodilators (nitric oxide and prostacyclin) and clotting inhibitors [thrombomodulin and tissue factor pathway inhibitor (TFPI)]. When the integrity of the endothelium is breached, vasoconstriction occurs through neural and chemical ...
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