Results 261 to 270 of about 1,985,012 (310)

Neurogenesis following brain ischemia

Developmental Brain Research, 2002
Following 5 or 10 min of global ischemia in the adult gerbil there is a tenfold increase in the birth of new cells in the subgranular zone of dentate gyrus of the hippocampus as assessed using BrdU incorporation. This begins at 7 days, peaks at 11 days, and decreases thereafter. Over the next month approximately 25% of the newborn cells disappear.
Frank R, Sharp, Jialing, Liu, Ramon, Bernabeu   +2 more
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Inflammation of the Brain after Ischemia

1996
Cytokines which promote emigration of leukocytes from the vascular lumen into the injured brain tissue are produced at the site of incipient cerebral infarction. The blood-borne invaders then accelerate the decomposition of brain cells by their toxic by-products, phagocytic action, and by the immune reaction.
K, Kogure, Y, Yamasaki, Y, Matsuo, H, Kato, H, Onodera   +4 more
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Catecholamines in Experimental Brain Ischemia

Archives of Neurology, 1975
Local cerebral ischemia was produced in rats by internal carotid artery injection of 35 mu carbon microspheres, and brain norepinephrine (NE), dopamine, and cyclic adenosine 3, 5-monophosphate (cAMP) were measured in embolized and intact hemispheres at intervals up to four hours. Sham-operated animals were controls.
K, Kogure, P, Scheinberg, A, Matsumoto, R, Busto, O M, Reinmuth   +4 more
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Vascular Protection in Brain Ischemia

Cerebrovascular Diseases, 2006
Vascular damage occurring after cerebral ischemia may lead to a worse outcome in patients with ischemic stroke, as it facilitates edema formation and hemorrhagic transformation. There are several phases in the development of vascular injury (acute, subacute and chronic) and different mediators act in each one.
Manuel, Rodríguez-Yáñez, Mar, Castellanos, Miguel, Blanco, Esther, Mosquera, José, Castillo   +4 more
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Global Brain Ischemia and Reperfusion

Annals of Emergency Medicine, 1996
Brain damage accompanying cardiac arrest and resuscitation is frequent and devastating. Neurons in the hippocampus CA1 and CA4 zones and cortical layers III and V are selectively vulnerable to death after injury by ischemia and reperfusion. Ultrastructural evidence indicates that most of the structural damage is associated with reperfusion, during ...
B C, White, L I, Grossman, B J, O'Neil, D J, DeGracia, R W, Neumar, J A, Rafols, G S, Krause   +6 more
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Thromboinflammation in Brain Ischemia

Stroke, 2022
Despite decades of promising preclinical validation and clinical translation, ischemic stroke still remains as one of the leading causes of death and disability worldwide. Within its complex pathophysiological signatures, thrombosis and inflammation, that is, thromboinflammation, are highly interconnected processes leading to cerebral vessel occlusion,
De Meyer, Simon F, Langhauser, Friederike, Haupeltshofer, Steffen, Kleinschnitz, Christoph, Casas, Ana I   +4 more
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Brain ischemia.

Topics in magnetic resonance imaging : TMRI, 1990
The sensitivity of MRI to changes in water state and volume makes it the most desirable modality for imaging early brain ischemia. Its sensitivity is reflected in the ability to show ischemic changes in the white matter of the asymptomatic elderly, which are attributed to axonal loss, demyelination, and gliosis.
C, Virapongse, E, Brown, J, Malat
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Cerebral ischemia and brain histamine

Brain Research Reviews, 2005
Cerebral ischemia induces excess release of glutamate and an increase in the intracellular Ca(2+) concentration in neurons, which provokes enzymatic process leading to irreversible neuronal injury. Histamine plays a role as a neurotransmitter in the mammalian brain, and histamine release from nerve endings is enhanced in ischemia by facilitation of ...
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Hyperthermia and Brain Ischemia

1997
This chapter reviews recent experimental studies concerning the detrimental consequences of hyperthermia on ischemic outcome. The chapter also discusses potential mechanisms underlying the detrimential consequences of mild hyperthermia. Fever is frequently observed following cardiopulmonary resuscitation and is considered prognostic of poor outcome in ...
W DALTONDIETRICH, R BUSTO
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Adenosine and Brain Ischemia

1995
Stroke is the second or third major cause of death in the “industrialized countries.” About 30% of patients die within a year after the first insult, and 20% remain dependent on care. So far there is no effective therapy available but one is desperately needed.
Karl A. Rudolphi, Peter Schubert
openaire   +1 more source