Results 261 to 270 of about 359,925 (305)
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Catecholamines in Experimental Brain Ischemia
Archives of Neurology, 1975Local cerebral ischemia was produced in rats by internal carotid artery injection of 35 mu carbon microspheres, and brain norepinephrine (NE), dopamine, and cyclic adenosine 3, 5-monophosphate (cAMP) were measured in embolized and intact hemispheres at intervals up to four hours. Sham-operated animals were controls.
K, Kogure +4 more
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Global Brain Ischemia and Reperfusion
Annals of Emergency Medicine, 1996Brain damage accompanying cardiac arrest and resuscitation is frequent and devastating. Neurons in the hippocampus CA1 and CA4 zones and cortical layers III and V are selectively vulnerable to death after injury by ischemia and reperfusion. Ultrastructural evidence indicates that most of the structural damage is associated with reperfusion, during ...
B C, White +6 more
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Topics in magnetic resonance imaging : TMRI, 1990
The sensitivity of MRI to changes in water state and volume makes it the most desirable modality for imaging early brain ischemia. Its sensitivity is reflected in the ability to show ischemic changes in the white matter of the asymptomatic elderly, which are attributed to axonal loss, demyelination, and gliosis.
C, Virapongse, E, Brown, J, Malat
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The sensitivity of MRI to changes in water state and volume makes it the most desirable modality for imaging early brain ischemia. Its sensitivity is reflected in the ability to show ischemic changes in the white matter of the asymptomatic elderly, which are attributed to axonal loss, demyelination, and gliosis.
C, Virapongse, E, Brown, J, Malat
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Cerebral ischemia and brain histamine
Brain Research Reviews, 2005Cerebral ischemia induces excess release of glutamate and an increase in the intracellular Ca(2+) concentration in neurons, which provokes enzymatic process leading to irreversible neuronal injury. Histamine plays a role as a neurotransmitter in the mammalian brain, and histamine release from nerve endings is enhanced in ischemia by facilitation of ...
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The pathophysiology of brain ischemia
Annals of Neurology, 1983AbstractBrain ischemia due to a critical reduction in cerebral blood flow is a well recognized and common cause of irreversible brain damage. The observation that brain cells are more resistant to ischemia than was previously assumed on the basis of clinical experience has stimulated considerable investigative work designed to determine those factors ...
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Hyperthermia and Brain Ischemia
1997This chapter reviews recent experimental studies concerning the detrimental consequences of hyperthermia on ischemic outcome. The chapter also discusses potential mechanisms underlying the detrimential consequences of mild hyperthermia. Fever is frequently observed following cardiopulmonary resuscitation and is considered prognostic of poor outcome in ...
W DALTONDIETRICH, R BUSTO
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1995
Stroke is the second or third major cause of death in the “industrialized countries.” About 30% of patients die within a year after the first insult, and 20% remain dependent on care. So far there is no effective therapy available but one is desperately needed.
Karl A. Rudolphi, Peter Schubert
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Stroke is the second or third major cause of death in the “industrialized countries.” About 30% of patients die within a year after the first insult, and 20% remain dependent on care. So far there is no effective therapy available but one is desperately needed.
Karl A. Rudolphi, Peter Schubert
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Cerebrovascular and brain metabolism reviews, 1993
Recent experimental data indicate a probable role of adenosine as an endogenous neuroprotective substance in brain ischemia. This nucleoside is rapidly formed during ischemia as a result of intracellular breakdown of ATP and it is subsequently transported into the extracellular space.
K A, Rudolphi +3 more
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Recent experimental data indicate a probable role of adenosine as an endogenous neuroprotective substance in brain ischemia. This nucleoside is rapidly formed during ischemia as a result of intracellular breakdown of ATP and it is subsequently transported into the extracellular space.
K A, Rudolphi +3 more
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Clinical Ischemia: Brain Infarcts
1977A cerebral infarct arises from a variety of causes either single or more commonly in combination of which three main factors seem to predominate: 1. cardiovascular insufficiency 2. local obstructions to blood flow in the “four principal brain arteries” and 3. changes within the blood itself.
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