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New England Journal of Medicine, 1995 Background. Bronchial hyperresponsiveness, a risk factor for asthma, consists of a heightened bronchoconstrictor response to a variety of stimuli. The condition has a heritable component and is closely related to serum IgE levels and airway inflammation.
Dirkje S Postma +2 more
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Epidemiology of bronchial hyperresponsiveness
Clinical Reviews in Allergy, 1989Asthma is a serious health problem, with both mortality and severity increasing in developed countries (55-57). The development of more specific tests to measure the prevalence and incidence of asthma is of paramount importance. Because measurement of BHR is the only practical objective method for assessing the abnormality of the airways in asthma, it ...
A J, Woolcock, J K, Peat
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Lymphocytes and bronchial hyperresponsiveness
Life Sciences, 1990Non-specific bronchial hyperresponsiveness can be defined as an increased responsiveness of the respiratory airways to physical, chemical and pharmacological stimuli. It is a characteristic feature of asthma. Knowledge of the mechanisms contributing to bronchial hyperresponsiveness can provide an insight into the pathogenesis of asthma and could lead ...
A J, Van Oosterhout, F P, Nijkamp
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Bronchial hyperresponsiveness today
Respiratory Medicine, 1993Patients with airflow limitation are currently classified into the clinical categories asthma and chronic obstructive pulmonary disease (COPD), based on symptoms, signs and abnormalities in function (1). One of the most prominent functional characteristics of patients with asthma (2) or COPD (3) is airway hyperresponsiveness (also called: bronchial ...
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Comprehensive Physiology, 1986
Abstract The sections in this article are: Bronchial Smooth Muscle Tension Regulation of Airways in Normal Subjects Regulation of Airways in Subjects With Bronchial Hyperresponsiveness
Ann J. Woolcock, Solbert Permutt
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Abstract The sections in this article are: Bronchial Smooth Muscle Tension Regulation of Airways in Normal Subjects Regulation of Airways in Subjects With Bronchial Hyperresponsiveness
Ann J. Woolcock, Solbert Permutt
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Nitric Oxide and Bronchial Hyperresponsiveness
2000Increasing evidence points to an important role for nitric oxide in the regulation of pulmonary functions and in pulmonary disease. In the respiratory tract, sensory nerves, endothelial cells, vascular and airway smooth muscle cells, inflammatory cells and the airway epithelium are sources of nitric oxide.
Nijkamp, F.P., Folkerts, G.
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Asymptomatic bronchial hyperresponsiveness in rhinitis
Journal of Allergy and Clinical Immunology, 1985Methacholine inhalation tests are used to help in the diagnosis of asthma when spirometry is normal. However, the significance of increased methacholine responsiveness in patients with rhinitis and no symptoms of asthma is not known. One possibility is that it is a false positive result; another possibility is that it indicates subclinical asthma.
E H, Ramsdale +3 more
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Cationic Proteins and Bronchial Hyperresponsiveness
American Journal of Respiratory Cell and Molecular Biology, 2000Chronic airway inflammation in asthma is characterized by the preponderance of eosinophils in the airway walls and lumens. Eosinophils are unique among granulocytes in that they contain intracytoplasmic granules, which stain with eosin. These granules contain a number of preformed enzymes and other proteins, including cationic proteins such as major ...
Madison, John M., Schramm, Craig M.
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Genetics of allergy and bronchial hyperresponsiveness
Clinical & Experimental Allergy, 1999Allergy and asthma are closely related complex diseases caused by a combination of both genetic and environmental influences. Two common genetic approaches, candidate gene studies and genome‐wide screens, have been used to localize and evaluate potential genetic factors that confer susceptibility or modify the phenotype of these diseases.
Howard, TD +5 more
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Role of Broxaterol in Bronchial Hyperresponsiveness
Respiration, 2009The protective effect of broxaterol against bronchial hyperresponsiveness has been assessed in several double-blind randomized controlled studies in stable asthmatic patients. In three cross-over studies, broxaterol (400 μg by metered dose inhaler) has been shown to significantly reduce methacholine responsiveness 30, 60 and 120 min after dosing.
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