Results 211 to 220 of about 193,403 (293)
Nanotoxicology in Caenorhabditis elegans
Prof. Dr. Dayong Wang
semanticscholar +1 more source
Microbial amyloids in neurodegenerative amyloid diseases
Numerous microbes, including those indigenous to the human microbiome, produce amyloidogenic proteins. These proteins have critical functions in bacterial physiology and their lifestyles. Emerging evidence indicates that microbial amyloids also interact directly or indirectly with host‐derived amyloid proteins, such as those which underlie Parkinson's ...
Timothy Sampson
wiley +1 more source
The mec-7 beta-tubulin gene of Caenorhabditis elegans is expressed primarily in the touch receptor neurons. [PDF]
Michel Hamelin+3 more
openalex +1 more source
The microbiota–gut–brain axis in Huntington's disease: pathogenic mechanisms and therapeutic targets
Disturbances of the microbiota–gut–brain axis are evident in people with Huntington's disease (HD) and in HD animal models. Here, we provide an overview of how the trillions of microorganisms residing in the gut, their secretory products, and the microbiota–gut–brain axis may contribute to HD pathogenesis.
Millicent N. Ekwudo+2 more
wiley +1 more source
The silencing of transposable elements is mediated by piRNAs through post‐transcriptional and transcriptional regulation. In Drosophila, piRNAs are processed within unique non‐membrane structures: nuage in germline cells and Yb bodies in somatic gonadal cells.
Ritsuko Suyama, Toshie Kai
wiley +1 more source
The primary structure of cytochrome c from the nematode Caenorhabditis elegans [PDF]
Jacques R. Vanfleteren+3 more
openalex +1 more source
Bacterial purine metabolism modulates C. elegans development and stress tolerance via DAF‐16
To investigate the impact of dietary components on animal growth, an Escherichia coli single‐gene deletion library was screened using the Caenorhabditis elegans growth model. Thirty‐four E. coli mutants were identified to delay worm development. Notably, E.
Min Feng+3 more
wiley +1 more source
Regulation of physiological and pathological condensates by molecular chaperones
Mounting evidence suggests that stress granules (SGs), dynamic membraneless compartments involved in cellular stress responses, can transition into pathological condensates upon improper disassembly. This review discusses the evidence supporting this notion.
Nadeen Akaree+5 more
wiley +1 more source
A simple and rapid screening method of TPA inhibitor with nematode Caenorhabditis elegans
Keiko Kurihara+3 more
openalex +1 more source