Results 11 to 20 of about 75,241 (308)

Calcium-channel blockers and anaesthesia [PDF]

open access: yesCanadian Journal of Anaesthesia, 1991
Verapamil was the first calcium-channel blocker (CCB). It has been used since 1962 in Europe then in Japan for its antiarrhythmic and coronary vasodilator effects. The CCB have become prominent cardiovascular drugs during the last 15 years. Many experimental and clinical studies have defined their mechanism of action, the effects of new drugs in this ...
Pierre Foëx   +2 more
openaire   +3 more sources

N-/T-Type vs. L-Type Calcium Channel Blocker in Treating Chronic Kidney Disease: A Systematic Review and Meta-Analysis

open access: yesPharmaceuticals, 2023
Renin-angiotensin system (RAS) inhibitors and calcium channel blockers (CCB) are often used together in chronic kidney disease (CKD). The PubMed, EMBASE, and Cochrane Library databases were searched to identify randomized controlled trials (RCTs) in ...
Mingming Zhao   +9 more
doaj   +1 more source

Calcium channel blockers

open access: yesMedicine, 2003
Overdose of calcium channel blockers is potentially lethal. Verapamil and diltiazem are much more dangerous than dihydropyridines. Hypotension with (verapamil and diltiazem) or without (dihydropyridines) heart block and bradycardia are the most important features.
Ian M Whyte   +5 more
openaire   +3 more sources

Calcium Channel Blocker Overdose Simulation

open access: yesMedEdPORTAL, 2015
Introduction Accidental poisoning and drug overdose are a common occurrence in the emergency medicine setting. Often it is unknown what substances the patient has taken.
Michael Maniaci   +2 more
doaj   +1 more source

Pre-injury beta blocker use does not affect the hyperdynamic response in older trauma patients

open access: yesJournal of Emergencies, Trauma and Shock, 2014
Purpose: Trauma dogma dictates that the physiologic response to injury is blunted by beta-blockers and other cardiac medications. We sought to determine how the pre-injury cardiac medication profile influences admission physiology and post-injury ...
David C Evans   +8 more
doaj   +1 more source

The fourth-generation Calcium channel blocker: Cilnidipine

open access: yesIndian Heart Journal, 2013
Several classes of antihypertensive agents have been in clinical use, including diuretics, α-blockers, β-blockers, angiotensin converting enzyme (ACE) inhibitors, angiotensin II type 1 receptor blockers (ARB), and organic calcium channel blockers (CCBs).
K. Sarat Chandra, G. Ramesh
doaj   +1 more source

Calcium Channel Blockers and Hypertension

open access: yesJournal of Cardiovascular Pharmacology and Therapeutics, 2014
Effective treatment of high blood pressure (BP) represents a key strategy for reducing the burden of hypertension-related cardiovascular and renal diseases. In spite of these well-established concepts, hypertension remains poorly controlled worldwide.
TOCCI, GIULIANO   +6 more
openaire   +4 more sources

Diltiazem for clozapine-induced generalized hyperhidrosis [PDF]

open access: yesMental Health Clinician, 2023
Background Clozapine can be associated with significant side effects and tolerability issues. Hyperhidrosis occurs less commonly and is unanticipated by clinicians because of clozapine's significant anticholinergic activity.
Kenton Emma M. PharmD   +2 more
doaj   +1 more source

Antiarrhythmic drug usage and prostate cancer: a population-based cohort study

open access: yesAsian Journal of Andrology, 2018
Even though the relationship between antiarrhythmic drug usage and subsequent prostate cancer (PCa) risk has recently been highlighted, relevant findings in the previous literature are still inconsistent.
Li-Ting Kao   +3 more
doaj   +1 more source

Calcium channel blockers and dementia [PDF]

open access: yesBritish Journal of Pharmacology, 2013
Degenerative dementia is mainly caused by Alzheimer's disease and/or cerebrovascular abnormalities. Disturbance of the intracellular calcium homeostasis is central to the pathophysiology of neurodegeneration. In Alzheimer's disease, enhanced calcium load may be brought about by extracellular accumulation of amyloid‐β.
Anne Eckert, V Nimmrich
openaire   +3 more sources

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