Results 261 to 270 of about 75,241 (308)
Some of the next articles are maybe not open access.

Calcium Channel Blockers and Atherosclerosis

Journal of Cardiovascular Pharmacology, 1990
There is evidence that calcium antagonists (calcium channel blockers) may suppress atheroma formation in animals fed high-fat diets. Studies on the antiatherosclerotic effects of calcium blockers have suggested a variety of possible mechanisms: (a) lowering of arterial pressure, (b) decrease in atherogenic plasma lipoproteins, (c) suppression of ...
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Chromatography of calcium channel blockers

Journal of Chromatography B: Biomedical Sciences and Applications, 1990
Numerous publications during the past ten years have described the determination of various calcium channel blockers in biological fluids, using gas and liquid chromatographic techniques. Diltiazem, verapamil, flunarizine and a growing number of dihydropyridines belong to this group of drugs, which in most instances are active at low plasma ...
Martin Ahnoff, Bengt-Arne Persson
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Calcium channel blockers in psychiatry

1996
The influence of calcium in mental disorders has been considered over time. For example, intravenous calcium gluconate was used for treatment of periodic psychosis as late as the 1970s. Also, mental symptoms may be prominent in patients with hypercalcemia of various causes.
Enrique S. Garza-Treviño   +1 more
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Calcium channel blocker toxicity

Annals of Emergency Medicine, 1990
A retrospective review was conducted of all patients who were reported to a regional poison control center after "overdose" of a calcium channel blocker during a two-year period (1987 and 1988). An analysis of 91 patient cases is presented after excluding allergic reactions, cases involving coingestants, and patients lost to follow-up.
Edward A. Ramoska   +2 more
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Calcium Channel Blockers [PDF]

open access: possible, 2015
Since its discovery more than 40 years ago, calcium channel blockers have been widely studied and used in cardiovascular medicine. Apart from their ability to reduce blood pressure they have been implemented in the treatment of various forms of angina pectoris (e.g.
Udo Sechtem, Peter Ong
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Calcium channel blockers: an update

The American Journal of Medicine, 2004
This paper reviews the current literature pertaining to calcium channel blockers, including their classification, properties, and therapeutic indications, in light of several recent trials that have addressed their safety. Calcium channel blockers are a structurally and functionally heterogeneous group of medications that are used widely to control ...
Alain N Bestawros   +2 more
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Calcium Channel Blocker Toxicity

Pediatric Emergency Care, 2009
Calcium channel blockers continue to be used for the management of a wide variety of adult and pediatric conditions including hypertension, angina pectoris, atrial arrhythmias, Raynaud phenomenon, and migraine headaches. With increased use comes increased potential for misuse and abuse.
Anna Maria Arroyo, Louise W. Kao
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Calcium Channel Blockers

2014
Calcium channel blockers (CCBs) can be pharmacologically classified into different subgroups as benzothiazepines (diltiazem), phenylalkylamines (verapamil, gallopamil), dihydropyridines (nifedipine, nicardipine, nisoldipine, nimodipine, nitradipine, nilvadipine, nitrendipine, benidipine, felodipine, amlodipine, barnidipine, cilnidipine, efonidipine ...
Esen Özkaya   +1 more
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Calcium channel blockers as tocolytics

Seminars in Perinatology, 2001
This article reviews the clinical and basic science investigations regarding the safety and efficacy of calcium channel blockers as tocolytic agents. The authors reviewed the English language literature on the pharmacology and clinical applications of calcium antagonists in obstetrics.
Alfred Z. Abuhamad   +2 more
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Calcium channel blockers and atherogenesis

The American Journal of Medicine, 1987
The events involved in atherogenesis include platelet deposition on damaged endothelial surfaces; migration and proliferation of smooth muscle cells; the formation of elastin, collagen, and glycosaminoglycans, followed by the penetration and complexing of lipoproteins; and, calcification.
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