Results 211 to 220 of about 88,833 (259)
Some of the next articles are maybe not open access.
Chromatography of calcium channel blockers
Journal of Chromatography B: Biomedical Sciences and Applications, 1990Numerous publications during the past ten years have described the determination of various calcium channel blockers in biological fluids, using gas and liquid chromatographic techniques. Diltiazem, verapamil, flunarizine and a growing number of dihydropyridines belong to this group of drugs, which in most instances are active at low plasma ...
M, Ahnoff, B A, Persson
openaire +2 more sources
Calcium channel blockers and cancer
The American Journal of Medicine, 2000We sought to explore the relation that has been previously reported between calcium channel blockers and an increased risk of cancer.We followed 3,511 participants, age 65 years or older, in the Duke Established Populations for Epidemiologic Studies of the Elderly for up to 10 years.
H J, Cohen +5 more
openaire +2 more sources
Calcium channel blockers as tocolytics
Seminars in Perinatology, 2001This article reviews the clinical and basic science investigations regarding the safety and efficacy of calcium channel blockers as tocolytic agents. The authors reviewed the English language literature on the pharmacology and clinical applications of calcium antagonists in obstetrics.
K E, Economy, A Z, Abuhamad
openaire +2 more sources
Calcium channel blocker toxicity
Annals of Emergency Medicine, 1990A retrospective review was conducted of all patients who were reported to a regional poison control center after "overdose" of a calcium channel blocker during a two-year period (1987 and 1988). An analysis of 91 patient cases is presented after excluding allergic reactions, cases involving coingestants, and patients lost to follow-up.
E A, Ramoska, H A, Spiller, A, Myers
openaire +2 more sources
Calcium channel blockers and atherogenesis
The American Journal of Medicine, 1987The events involved in atherogenesis include platelet deposition on damaged endothelial surfaces; migration and proliferation of smooth muscle cells; the formation of elastin, collagen, and glycosaminoglycans, followed by the penetration and complexing of lipoproteins; and, calcification.
openaire +2 more sources
Calcium channel blockers and asthma
Lung, 1986An increase in cytoplasmic Ca++ concentration can activate not only respiratory smooth muscles, but also mast cells, bronchial mucus glands, and the vagi. Altered control of cytoplasmic Ca++ may also be related to the development of bronchial hyperreactivity.
So, SY, Ip, M, Lam, WK
openaire +4 more sources
Review of Calcium-Channel Blockers
The Nurse Practitioner, 1986Calcium-channel blocking drugs were introduced to the United States in 1982. The three approved calcium-channel blockers--nifedipine, verapamil and diltiazem--have offered new treatments for angina. This article presents an overview of these drugs with emphasis on their mechanisms of action, clinical use and guidelines for the nurse practitioner in ...
openaire +2 more sources
Calcium channel blockers and stroke.
Aging clinical and experimental research, 2005The most frequent indication for Calcium Channel Blockers (CCBs) is their use as antihypertensive agents for primary or secondary stroke prevention. Hypertension contributes to virtually all mechanisms of stroke, of atherothrombotic, lacunar, cardioembolic and intraparenchymal hemorrhage types.
INZITARI, DOMENICO, POGGESI, ANNA
openaire +2 more sources
Calcium Channel Blockers and Atherosclerosis
Journal of Cardiovascular Pharmacology, 1990There is evidence that calcium antagonists (calcium channel blockers) may suppress atheroma formation in animals fed high-fat diets. Studies on the antiatherosclerotic effects of calcium blockers have suggested a variety of possible mechanisms: (a) lowering of arterial pressure, (b) decrease in atherogenic plasma lipoproteins, (c) suppression of ...
openaire +2 more sources
Calcium channel blocker overdose
The American Journal of Emergency Medicine, 1995A case of diltiazem overdose with significant hemodynamic compromise is presented. Multiple therapeutic modalities were attempted with limited results. Control was finally achieved with a combination of norepinephrine, dobutamine, and cardiac pacing.
L, Proano, W K, Chiang, R Y, Wang
openaire +2 more sources