Results 181 to 190 of about 690,914 (230)

Drug dosage protocol for calcium oxalate stone

Urological Research, 2009
In earlier studies, we have confirmed that in most patients with calcium oxalate stone formation, a combination of allopurinol and pyridoxine is best suited for treatment and prevention of the stone forming process. The objective of this study is to identify the most effective directed medical treatment of urinary stones.
Y M Fazil, Marickar, Abiya, Salim
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Calcium oxalate stone and gout

Urological Research, 2009
Gout is well known to be produced by increased uric acid level in blood. The objective of this paper is to assess the relationship between gout and calcium oxalate stone formation in the humans. 48 patients with combination of gout and calcium oxalate stone problem were included.
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Intracellular Events in the Initiation of Calcium Oxalate Stones

Nephron Experimental Nephrology, 2004
This review summarizes our current understanding of intracellular events in the initiation of kidney stone formation, focusing on results from studies using renal epithelial cells in vitro. Such studies have shown that oxalate – either in crystalline or in soluble form – triggers a spectrum of responses in renal cells that favor stone formation ...
Julie A, Jonassen, Lu-Cheng, Cao, Thomas, Honeyman, Cheryl R, Scheid   +3 more
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Significance of Glycosaminoglycans for the Formation of Calcium Oxalate Stones

American Journal of Kidney Diseases, 1991
Glycosaminoglycans (GAG) are polysaccharide chains composed of repeating disaccharides of identical composition. Little is known about the mechanism of their excretion, but there is no doubt that urinary GAGs are degradation products of high molecular weight proteoglycans.
A, Hesse, H, Wuzel, W, Vahlensieck
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Observations on Calcium Oxalate Stone Formers

British Journal of Urology, 1992
Summary Biochemical studies were performed on 22 adult patients with idiopathic recurrent calcium oxalate renal stone disease and 23 healthy controls. It was found that urinary glutamic‐oxaloacetic acid transaminase (UGOT) and urinary glutamic‐pyruvic acid transaminase (UGPT) activity was low and lactate ...
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Genes in idiopathic calcium oxalate stone disease

World Journal of Urology, 1997
An examination of the urinary excretions of 101 normal subjects indicated that the major genetic influence on calcium excretion is a codominant pair of alleles giving rise to three phenotypes, low, intermediate and high (hypercalciuric) excretors. This inference was based on variance, Hardy-Weinberg and segregation analyses.
H O, Goodman, R, Brommage, D G, Assimos, R P, Holmes   +3 more
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Microorganisms and Calcium Oxalate Stone Disease

Nephron Physiology, 2004
Microorganisms may have a role in the pathogenesis and prevention of kidney stones. The subjects of this review include nanobacteria, <i>Oxalobacter formigenes</i>, and lactic acid bacteria. Not reviewed here is the well-described role of infections of the urinary tract with <i>Proteus</i> species and other urease-producing ...
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Submicroscopic Investigations on Calcium Oxalate Stone Genesis

European Urology, 1979
Scanning electron microscopy of calcium oxalate stones indicates a zonary structure of the Weddellite crystals of urine concrements which is due to concentration-dependent growth oscillations representing the precipitation of various calcium oxalate hydrates.
W, Berg, P, Lange, C, Bothor, D, Rössler   +3 more
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Genetic Factors in Calcium Oxalate Stone Disease

Journal of Urology, 1995
Idiopathic calcium oxalate nephrolithiasis is a complex, multifactorial disease resulting from an interaction between environmental and genetic factors. Urinary characteristics often associated with the disease include hypercalciuria, hyperoxaluria, hypocitraturia and hyperuricosuria. Roles have also been suggested for glycosaminoglycans, magnesium and
H O, Goodman, R P, Holmes, D G, Assimos
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Studies on crystalluria in calcium oxalate stone formers

Urological Research, 1984
The excretion of calcium oxalate and calcium phosphate crystals was studied in fractionated 24 h urine from 7 men with recurrent calcium oxalate stone disease, both before and during daily administration of 5 mg bendroflumethiazide. Urinary calcium, oxalate, magnesium, citrate, phosphate, pH, and inhibition of calcium oxalate crystal growth rate were ...
C, Ahlstrand, H G, Tiselius, L, Larsson
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