RETRACTION: Beta3-Adrenergic Receptor Activation Alleviates Cardiac Dysfunction in Cardiac Hypertrophy by Regulating Oxidative Stress. [PDF]
Cellular Longevity OMA.
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IS39, a novel non‐peptide PKR1 agonist, confers cardioprotection against doxorubicin‐induced toxicity. IS39 activates PKR1‐mediated pro‐survival signalling in cardiomyocytes, reducing reactive oxygen species (ROS), DNA damage and fibrosis markers. In vivo, IS39 preserves cardiac geometry and function in mice exposed to chronic doxorubicin challenge ...
Anais Audebrand +8 more
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A circular RNA derived from the ryanodine receptor 2 locus controls cardiac hypertrophy and calcium handling. [PDF]
Pan W +17 more
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Myeloid MyD88 Mediates Macrophage Infiltration and Activation in Ang II-Induced Cardiac Hypertrophy. [PDF]
Lin K +6 more
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Activation of FoxO1 prevents and reverses cardiac hypertrophy from diverse stimuli. [PDF]
Martin TG +4 more
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Corrigendum to "Wogonin Inhibits Cardiac Hypertrophy by Activating Nrf-2-Mediated Antioxidant Responses". [PDF]
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Special Issue "Cellular and Molecular Biology of Cardiac Hypertrophy and Heart Failure: Pathogenesis, Diagnostics and Treatment". [PDF]
Karmazyn M.
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Protosappanin A protects against pathological cardiac hypertrophy by inhibiting oxidative stress and NLRP3 inflammasome-mediated pyroptosis via activation of the Nrf2 signaling pathway. [PDF]
He Q, Zheng Y, Yan X, Lv S, Yu B.
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Experimental Cardiac Hypertrophy
New England Journal of Medicine, 1970STUDIES on the process by which enlargement of the heart occurs1 2 3 4 have been aided by developments in molecular biology.
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Endothelin and Cardiac Hypertrophy
Life Sciences, 1997Cardiac hypertrophy is a commonly observed complication of a variety of cardiovascular diseases. As well as mechanical stresses such as pressure overload, several humoral factors may contribute to the development of cardiac hypertrophy. In the early 1990s, endothelin-1 (ET-1) was found to induce cardiomyocyte hypertrophy in cultured neonatal rat ...
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