Results 281 to 290 of about 16,339,920 (405)

Bioinformatics analysis of immune infiltration and cell cycle hub genes in atopic dermatitis. [PDF]

open access: yesBiochem Biophys Rep
Liu X   +8 more
europepmc   +1 more source

Cell-Cycle Regulation Accounts for Variability in Ki-67 Expression Levels.

open access: yesCancer Research, 2017
M. Sobecki   +7 more
semanticscholar   +1 more source

Not just a by‐product: circular DNA molecules derived from V(D)J recombination are linked to worse prognosis in B‐cell leukemia

open access: yesMolecular Oncology, EarlyView.
Gao et al. report that circular DNA molecules created as by‐products of V(D)J recombination during lymphocyte maturation (ESCs) can replicate and be retained for much longer than previously thought in healthy cells. In BCP‐ALL cells, increased ESC abundance correlates with a greater chance of relapse likely mediated by their ability to induce genome ...
Davide Pradella, Andrea Ventura
wiley   +1 more source

Aggressive prostate cancer is associated with pericyte dysfunction

open access: yesMolecular Oncology, EarlyView.
Tumor‐produced TGF‐β drives pericyte dysfunction in prostate cancer. This dysfunction is characterized by downregulation of some canonical pericyte markers (i.e., DES, CSPG4, and ACTA2) while maintaining the expression of others (i.e., PDGFRB, NOTCH3, and RGS5).
Anabel Martinez‐Romero   +11 more
wiley   +1 more source

Protein arginine methyltransferase 7 regulates the cell cycle and promotes the progression of prostate cancer. [PDF]

open access: yesOncol Lett
Wan H   +11 more
europepmc   +1 more source

Non-canonical functions of cell cycle cyclins and cyclin-dependent kinases

open access: yesNature reviews. Molecular cell biology, 2016
P. Hydbring   +2 more
semanticscholar   +1 more source

The neural crest‐associated gene ERRFI1 is involved in melanoma progression and resistance toward targeted therapy

open access: yesMolecular Oncology, EarlyView.
ERRFI1, a neural crest (NC)‐associated gene, was upregulated in melanoma and negatively correlated with the expression of melanocytic differentiation markers and the susceptibility of melanoma cells toward BRAF inhibitors (BRAFi). Knocking down ERRFI1 significantly increased the sensitivity of melanoma cells to BRAFi.
Nina Wang   +8 more
wiley   +1 more source

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