Results 281 to 290 of about 8,365,967 (315)
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Molecular Microbiology, 1998
Sequence similarity data suggest that archaeal chromosome replication is eukaryotic in character. Putative nucleoid‐processing proteins display similarities to both eukaryotic and bacterial counterparts, whereas cell division may occur through a predominantly bacterial mechanism.
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Sequence similarity data suggest that archaeal chromosome replication is eukaryotic in character. Putative nucleoid‐processing proteins display similarities to both eukaryotic and bacterial counterparts, whereas cell division may occur through a predominantly bacterial mechanism.
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Philosophical Transactions of the Royal Society of London. Series B: Biological Sciences, 1995
Abstract Cell proliferation involves duplication of all cell constituents and their more-or-less equal segregation to daughter cells. It seems probable that the performance of primitive cell-like structures would have been dogged by poor duplication and segregation fidelity, and by parasitism. This favoured evolution of the genome and
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Abstract Cell proliferation involves duplication of all cell constituents and their more-or-less equal segregation to daughter cells. It seems probable that the performance of primitive cell-like structures would have been dogged by poor duplication and segregation fidelity, and by parasitism. This favoured evolution of the genome and
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Current Opinion in Cell Biology, 1991
This review focuses on the G1 regulation of the p34cdc2/CDC28 kinase by cyclin-like proteins, which has substantially altered our understanding of cell cycle control. We discuss advances in elucidating the molecular composition of the mitotic apparatus, an essential step in understanding its cell-cycle-dependent assembly and functions.
M G, Goebl, M, Winey
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This review focuses on the G1 regulation of the p34cdc2/CDC28 kinase by cyclin-like proteins, which has substantially altered our understanding of cell cycle control. We discuss advances in elucidating the molecular composition of the mitotic apparatus, an essential step in understanding its cell-cycle-dependent assembly and functions.
M G, Goebl, M, Winey
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Annual Review of Plant Biology, 2003
Cell division in plants is controlled by the activity of cyclin-dependent kinase (CDK) complexes. Although this basic mechanism is conserved with all other eukaryotes, plants show novel features of cell-cycle control in the molecules involved and their regulation, including novel CDKs showing strong transcriptional regulation in mitosis.
Walter, Dewitte, James A H, Murray
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Cell division in plants is controlled by the activity of cyclin-dependent kinase (CDK) complexes. Although this basic mechanism is conserved with all other eukaryotes, plants show novel features of cell-cycle control in the molecules involved and their regulation, including novel CDKs showing strong transcriptional regulation in mitosis.
Walter, Dewitte, James A H, Murray
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International Journal of Radiation Biology and Related Studies in Physics, Chemistry and Medicine, 1986
The control of cell proliferation, in physiological terms, depends not so much on our understanding of the sequence of biochemical events unfolding as a cell progresses through its proliferation cycle, as upon the recognition by a tissue of the demands for functional cells of a particular type. After considering the modes of control possible, i.e.
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The control of cell proliferation, in physiological terms, depends not so much on our understanding of the sequence of biochemical events unfolding as a cell progresses through its proliferation cycle, as upon the recognition by a tissue of the demands for functional cells of a particular type. After considering the modes of control possible, i.e.
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Science, 1996
Uncontrolled cell proliferation is the hallmark of cancer, and tumor cells have typically acquired damage to genes that directly regulate their cell cycles. Genetic alterations affecting p16 INK4a and cyclin D1, proteins that govern phosphorylation of the retinoblastoma protein (RB) and ...
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Uncontrolled cell proliferation is the hallmark of cancer, and tumor cells have typically acquired damage to genes that directly regulate their cell cycles. Genetic alterations affecting p16 INK4a and cyclin D1, proteins that govern phosphorylation of the retinoblastoma protein (RB) and ...
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