Results 41 to 50 of about 48,699 (150)

Turner syndrome and postpubertal Empty sella syndrome: a case report and literature review

open access: yesFrontiers in Endocrinology
IntroductionTurner syndrome is a common sex chromosome disorder characterized by short stature, gonadal dysgenesis, and hypergonadotropic hypogonadism.
Fanyu Lin   +3 more
doaj   +1 more source

Case series of unusual cases of hypothyroidism-induced pericardial effusion

open access: yesJournal of Family Medicine and Primary Care
Hypothyroidism is an endocrine disorder with a high worldwide prevalence and diverse clinical presentation and can affect multiple organ systems. It can be asymptomatic and subclinical or overtly symptomatic and can prove to be fatal if left untreated ...
Ratnakar Sahoo   +3 more
doaj   +1 more source

Dynamic changes of central thyroid functions in the management of Cushing's syndrome

open access: yesArchives of Endocrinology and Metabolism, 2018
Objective The aim of this study was to determine the frequency of central thyroid dysfunctions in Cushing's syndrome (CS). We also aimed to evaluate the frequency of hyperthyroidism due to the syndrome of the inappropriate secretion of TSH (SITSH), which
Sema Ciftci Dogansen   +4 more
doaj   +1 more source

Severe fatigue due to valproate-induced hypothyroidism in a case of bipolar disorder

open access: yesAnnals of General Psychiatry, 2020
Background Valproate-induced hypothyroidism is a rare condition and has been considered asymptomatic. Here, we report a case of bipolar I disorder who developed symptomatic valproate-induced hypothyroidism.
Tadashi Kanamori   +5 more
doaj   +1 more source

The Clinical Spectrum of Resistance to Thyroid Hormone Alpha in Children and Adults

open access: yesJCRPE, 2021
Resistance to thyroid hormone alpha occurs due to pathogenic, heterozygous variants in THRA. The entity was first described in 2012 and to date only a small number of patients with varying severity have been reported.
İbrahim Mert Erbaş, Korcan Demir
doaj   +1 more source

The Changes of Thyroid Function and Related Factors in Critical Patients without Thyroid Illness in ICU: A Retrospective Cross-Sectional Study

open access: yesTherapeutics and Clinical Risk Management, 2022
Jiang-Nan Zhang, Xi-Le Zhao Department of Endocrinology, The First Hospital of Fuzhou, Fuzhou, 350009, People’s Republic of ChinaCorrespondence: Jiang-Nan Zhang, Department of Endocrinology, The First Hospital of Fuzhou, No. 190 of DaDao Street, Taijiang
Zhang JN, Zhao XL
doaj  

Subclinical hypothyroidism or central hypothyroidism—The danger of thyroid function misinterpretation

open access: yesClinical Case Reports, 2018
Key Clinical Message Correct interpretation of thyroid function tests is critical to providing appropriate care to patients with suspected thyroid disease.
Oluwaseun Anyiam   +2 more
doaj   +1 more source

Hypoprolactinemia as a Clue to Diagnosis of Mild Central Hypothyroidism due to IGSF1 Deficiency

open access: yesJCRPE, 2020
Loss-of-function mutations of IGSF1 are an X-linked cause of central hypothyroidism (CeH) and hypoprolactinemia. A boy who is now 15.2 years old presented at the age of 7.69 years for evaluation of obesity.
Anastasios Papadimitriou   +4 more
doaj   +1 more source

Mitotane-Induced Hypothyroidism and Dyslipidemia in Adrenocortical Carcinoma: Sex Differences and Novel Evidence from a Thyroid Cell Model

open access: yesCurrent Oncology
Adrenocortical carcinoma (ACC) is a rare and aggressive cancer with limited treatment options, commonly managed with mitotane, which can cause serious side effects, including central hypothyroidism and dyslipidemia.
Irene Tizianel   +9 more
doaj   +1 more source

Novel MTTP Gene Mutation in a Case of Abetalipoproteinemia with Central Hypothyroidism

open access: yesJCRPE, 2020
Abetalipoproteinaemia (ABL) is an autosomal recessive disorder characterized by very low plasma concentrations of total cholesterol and triglyceride (TG). It results from mutations in the gene encoding microsomal TG transfer protein (MTTP).
Pembe Soylu Ustkoyuncu   +6 more
doaj   +1 more source

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