Results 251 to 260 of about 624,789 (324)

Transient cerebellum-related network disruption characterizes postoperative cognitive changes after dominant hemisphere subtemporal meningioma resection. [PDF]

Acta Neurochir (Wien)
Liu Z, Li Y, Jiang J, Xu J, Liu Y, Zhang J, Wu Z, Li S, Wang L.   +8 more
europepmc   +1 more source

Heparan Sulfate Proteoglycans: Master Regulators of Cellular Signaling, Tissue Development, and Neural Function

Journal of Neuroscience Research, Volume 104, Issue 9, September 2026.
Ubiquitous cell‐associated, cytoplasmic, synaptic and extracellular matrix HS‐proteoglycans are multifunctional regulators of tissue form and function. HS‐proteoglycans have diverse functions as growth factor co‐receptors in cell proliferation, differentiation, tissue development, mechanical stabilization, synaptogenesis, synaptic plasticity, and ...
James Melrose
wiley   +1 more source

Recurrent multiloculated cystic variant of Lhermitte-Duclos disease managed with cyst fenestration and Ommaya reservoir placement: illustrative case. [PDF]

J Neurosurg Case Lessons
Uchida D, Hiu T, Masui K, Yamaguchi R, Nakamura H, Takeuchi M, Shirahama M, Ogawa Y, Matsuo A, Matsunaga Y, Baba S, Ujifuku K, Yoshida K, Morofuji Y, Matsuo T.   +14 more
europepmc   +1 more source

Cerebellar cryptococcoma simulating metastatic neoplasm Criptococoma cerebelar simulando neoplasia metastática

Arquivos de Neuro-Psiquiatria, 2009
Valter José Sillero-Filho, Arthur Borges Martins de Souza, Rafael Pereira Vaitsman, Rodrigo Ayres de Morais, Pedro de Oliveira Coutinho, Renata Lengruber, José Massoud Salame, José Fernando Guedes-Corrêa, Paulo Roberto Louzada   +8 more
openaire   +1 more source

Mitochondria as the Hub of Apoptosis: A Comprehensive Insight From Mitochondria to Interactions With Other Organelles

MedComm, Volume 7, Issue 9, September 2026.
Mitochondria is the hub of apoptosis in various diseases. The disruption of mitochondrial structure (including membrane rupture, cristae remodeling, and mitochondrial membrane lipid redistribution), the imbalance of mitochondrial dynamics (including fusion and fission, autophagy), the release, disruption, and mutation of mitochondria DNA, as well as ...
Rubin Tan, Jinming Zhao, Rong Wang, Yue Shi, Zaozao Chen, Yixuan Ma, Na Li, Wenqing Duan, Duo Lin, Chunxiang Zhang   +9 more
wiley   +1 more source

Gliosarcoma of the right cerebellar hemisphere and parahippocampal region: A case report and literature review. [PDF]

Oncol Lett
Wu F, Yang Y, Wang Z, Jiang R, Wu T, Yu T, Wang P.   +6 more
europepmc   +1 more source

Cell Death in Neurodegenerative Diseases: Molecular Mechanisms and Therapeutic Targets

MedComm, Volume 7, Issue 9, September 2026.
Regulated cell death pathways, including apoptosis, necroptosis, pyroptosis, ferroptosis, and autophagy‐dependent cell death, interact with mitochondrial dysfunction, proteostasis failure, lysosomal stress, glial remodeling, and neuroinflammation across major neurodegenerative diseases.
Tianjiao Li, Qian Zhang, Yichen Wu, Ruixue Huang   +3 more
wiley   +1 more source

Paraneoplastic neurologic syndrome in a 65-year-old woman with small cell lung cancer. [PDF]

CMAJ
Hiraoka Y, Hosoi Y, Yasumi K, Ozawa Y.
europepmc   +1 more source

Advances in FGF/FGFR Signaling: Implications for Disease and Therapy

MedComm, Volume 7, Issue 9, September 2026.
The FGF/FGFR signaling is indispensable for the maintenance of physiological homeostasis and governs multiple biological processes, including embryonic development, bone metabolism, angiogenesis, and neurogenesis, whereas aberrant hyperactivation of this pathway drives the progression of malignancies and autoimmune disorders, including inflammatory ...
Miaoyu Song, Xi Liu, Yang Xiao, Yongsheng Li, Huakan Zhao   +4 more
wiley   +1 more source

Severe cerebellitis as a manifestation of infliximab-associated overlap of neurosarcoidosis and myelin oligodendrocyte glycoprotein antibody-associated disease - case report. [PDF]

Front Immunol
Hütter G, Winkelhausen M, Tönges L, Gold R, Faissner S.   +4 more
europepmc   +1 more source