Results 171 to 180 of about 98,361 (208)
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1969
1. New biochemical results have shown that — in accordance with electron microscopical investigations — the edema of the cerebral cortex is completely different from the edema of the white substance, because of its intracellular localization.
K. Schürmann +4 more
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1. New biochemical results have shown that — in accordance with electron microscopical investigations — the edema of the cerebral cortex is completely different from the edema of the white substance, because of its intracellular localization.
K. Schürmann +4 more
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Pediatrics in Review, 1983
Although no new drugs have become available for improved treatment of cerebral edema, the increased understanding of the pathophysiology of the edematous process has led to more rational approaches to therapy, both in patients with cytotoxic intracellular brain edema and those with extracellular vasogenic brain edema.
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Although no new drugs have become available for improved treatment of cerebral edema, the increased understanding of the pathophysiology of the edematous process has led to more rational approaches to therapy, both in patients with cytotoxic intracellular brain edema and those with extracellular vasogenic brain edema.
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Cerebral Swelling and Cerebral Edema
Journal of Neuropathology & Experimental Neurology, 1952Robert G. Small, W. A. Krehl
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Cerebral Edema and Neurointensive Care
Pediatric Clinics of North America, 1980Cerebral edema may be vasogenic or cytoxic, and its main significance is raised intracranial pressure. Vasogenic edema results from damage to the blood vessel wall. The hallmark of cytotoxic edema is intracellular accumulation of fluid which occurs because of the exposure of the cells of the brain to toxic substances. Few diagnostic tests will document
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1982
In many not necessarily fatal brain diseases concomitant cerebral edema may cause the death of the patient. Even extracerebral noxae such as hypoxia, substrate deficit due to disturbance of respiration, circulation or metabolism and intoxications may initiate critical cerebral edema (Table 1).
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In many not necessarily fatal brain diseases concomitant cerebral edema may cause the death of the patient. Even extracerebral noxae such as hypoxia, substrate deficit due to disturbance of respiration, circulation or metabolism and intoxications may initiate critical cerebral edema (Table 1).
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Losing the dogmatic view of cerebral autoregulation
Physiological Reports, 2021Patrice Brassard +2 more
exaly
Hepatic Encephalopathy and Cerebral Edema
Seminars in Liver Disease, 1986R J Ede, Roger Williams
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