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USP43 promotes cerebral ischemia-reperfusion injury via activation of TAK1. [PDF]

Cell Biosci
Dong Y, Hu S, Zhang R, Zhang T, Feng Y, Sun W, Zhang P, Chen J, Zhao W.   +8 more
europepmc   +1 more source

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Experimental cerebral ischemia

Neurosurgery, 1980
The basic concepts of the pathology and pathophysiology of cerebral ischemia are presented. Special emphasis is given to the changes that occur in the microcirculation and in autoregulation because of their primary role in the pathogenesis of cerebral infarction. The effects of adrenal steroids, barbiturates, and cerebral revascularization are reviewed.
F G, Diaz, J I, Ausman
openaire   +2 more sources

MicroRNAs in Cerebral Ischemia

Translational Stroke Research, 2010
Pathogenesis of cerebral ischemia has so far been described in the context of proteins and the pathways that they regulate. The discovery of biomarkers has also been focussed mainly on proteins and to some extent on the mRNAs that encode them. The knowledge on the role of microRNAs in understanding the pathogenesis of cerebral ischemia is still at its ...
Lim, K.-Y., Chua, J.-H., Tan, J.-R., Swaminathan, P., Sepramaniam, S., Armugam, A., Wong, P.T.-H., Jeyaseelan, K.   +7 more
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CEREBRAL ISCHEMIA AND INFARCTION

Neuroimaging Clinics of North America, 1994
Cerebral infarction is most commonly related to atherosclerotic disease in the carotid and vertebrobasilar circulations. TIAs are manifestations of this same disease process and may occur before a complete infarction. The transformation of pale to hemorrhagic infarction may result from reperfusion of an infarct or from migration of an embolus.
A N, Hasso, W A, Stringer, K D, Brown
openaire   +2 more sources