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Microvascular Research, 1977
Abstract Total cerebral ischemia often injures or alters the microcirculation. This injury is highly variable within microregions, often resulting in different patterns of reperfusion within 100 μm. There is a growing consensus that the microcirculation injury is a consequence of ischemic brain injury rather than an essential pathogenetic step to it.
Wilhelm Erdmann +3 more
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Abstract Total cerebral ischemia often injures or alters the microcirculation. This injury is highly variable within microregions, often resulting in different patterns of reperfusion within 100 μm. There is a growing consensus that the microcirculation injury is a consequence of ischemic brain injury rather than an essential pathogenetic step to it.
Wilhelm Erdmann +3 more
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Archives of Surgery, 1970
Eighteen of twenty patients with subclavian or innominate artery obstruction experienced episodes of transient cerebral arterial insufficiency; one patient suffered acute stroke and coma; all patients had angiographic evidence of subclavian or innominate artery obstruction; and most were shown to have retrograde flow in a vertebral artery. Two clinical
Theodore Drapanas +2 more
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Eighteen of twenty patients with subclavian or innominate artery obstruction experienced episodes of transient cerebral arterial insufficiency; one patient suffered acute stroke and coma; all patients had angiographic evidence of subclavian or innominate artery obstruction; and most were shown to have retrograde flow in a vertebral artery. Two clinical
Theodore Drapanas +2 more
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JAMA, 1961
Because of the sensitivity of the brain to hypoxia, acute cerebral ischemia due to operative or postoperative disruption of circulatory homeostasis, especially in older persons, should not be surprising. Six cases were selected to illustrate various peripheral circulatory mechanisms which, in conjunction with unrecognized preexisting cerebral vascular ...
Joseph F. Fazekas +5 more
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Because of the sensitivity of the brain to hypoxia, acute cerebral ischemia due to operative or postoperative disruption of circulatory homeostasis, especially in older persons, should not be surprising. Six cases were selected to illustrate various peripheral circulatory mechanisms which, in conjunction with unrecognized preexisting cerebral vascular ...
Joseph F. Fazekas +5 more
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Hemichannels in Cerebral Ischemia
Current Molecular Medicine, 2009Hemichannels are transmembrane channels that represent the functional subunits of gap junctions. Each hemichannel is composed of a connexin or pannexin hexamer and, after being transported to the membrane, remains unpaired until it is incorporated in a gap junction. Several studies have already provided evidence that gap junction-mediated intercellular
Markus Schwaninger +2 more
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2011
Rodent models of focal cerebral ischemia have been extremely useful in elucidating pathomechanisms of human stroke. Most commonly, a monofilament is advanced through the internal carotid artery of rodents to occlude the origin of the middle cerebral artery thus leading to critical ischemia in the corresponding vascular territory.
Guido Stoll +3 more
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Rodent models of focal cerebral ischemia have been extremely useful in elucidating pathomechanisms of human stroke. Most commonly, a monofilament is advanced through the internal carotid artery of rodents to occlude the origin of the middle cerebral artery thus leading to critical ischemia in the corresponding vascular territory.
Guido Stoll +3 more
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CEREBRAL ISCHEMIA AND INFARCTION
Neuroimaging Clinics of North America, 1994Cerebral infarction is most commonly related to atherosclerotic disease in the carotid and vertebrobasilar circulations. TIAs are manifestations of this same disease process and may occur before a complete infarction. The transformation of pale to hemorrhagic infarction may result from reperfusion of an infarct or from migration of an embolus.
A N, Hasso, W A, Stringer, K D, Brown
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Focal Cerebral Ischemia and Migraine
Cephalalgia, 1985Consistent literature data on the cerebrovascular risk in migraineous patients are lacking. Available preliminary clinical data (Italian Cooperative Cross-Sectional Case-Control Study) suggest that migraine can hardly be considered a relevant pathogenetic associated risk factor of focal cerebral ischemia.
PRENCIPE M +3 more
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Electrophysiology of cerebral ischemia
Neuropharmacology, 2008Organized brain activity requires the coordinated firing of vast numbers of nerve cells. To maintain this, all these cells must be adequately polarized, their axons capable of conducting action potentials and releasing transmitters at an even greater numbers of synapses. Hence the often dire consequences of any interruption in the normal supply of O(2)
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Therapeutics for Focal Cerebral Ischemia
Neurosurgery, 1980A review of the therapeutic modalities available for the treatment of focal cerebral ischemia is presented. The theoretical aspects and present practical applications of each treatment are discussed. The possibilities for future research and for the implementation of new modalities are indicated.
Warren R. Selman, Robert F. Spetzler
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Polyamines and cerebral ischemia
1998It has been well established that alterations in polyamine metabolism are associated with animal models of global ischemia. Recently, this has been extended to include models of focal ischemia and traumatic brain injury. There is much evidence to support the idea that polyamines may play a multifaceted detrimental role following ischemia reperfusion ...
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