Results 211 to 220 of about 38,798 (242)
MiRNA expression profiling reveals a potential role of microRNA-148b-3p in cerebral vasospasm in subarachnoid hemorrhage. [PDF]
Sci RepRyu JY +11 more
europepmc +1 more source
Intracranial dynamics biomarkers at traumatic cerebral vasospasm. [PDF]
Brain SpineTrofimov AO +8 more
europepmc +1 more source
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Journal of Neuroscience Nursing, 1980
Because previous studies suggest that prostaglandins (PGs) are involved in the contraction of cerebral arteries, the present study was undertaken to assess the direct effects of arachidonic acid, the precursor of PGs, on isolated dog basilar arteries and to ascertain the actions of three prostaglandin synthesis inhibitors, aspirin, indomethacin, and ...
M K, Gerk, N F, Kassell
+6 more sources
Because previous studies suggest that prostaglandins (PGs) are involved in the contraction of cerebral arteries, the present study was undertaken to assess the direct effects of arachidonic acid, the precursor of PGs, on isolated dog basilar arteries and to ascertain the actions of three prostaglandin synthesis inhibitors, aspirin, indomethacin, and ...
M K, Gerk, N F, Kassell
+6 more sources
Neurosurgery Clinics of North America, 2014
Cerebral vasospasm causes delayed ischemic neurologic deficits after aneurysmal subarachnoid hemorrhage. This is a well-established clinical entity with significant associated morbidity and mortality. The underlying patholphysiology is highly complex and poorly understood.
Christopher D, Baggott +1 more
openaire +2 more sources
Cerebral vasospasm causes delayed ischemic neurologic deficits after aneurysmal subarachnoid hemorrhage. This is a well-established clinical entity with significant associated morbidity and mortality. The underlying patholphysiology is highly complex and poorly understood.
Christopher D, Baggott +1 more
openaire +2 more sources
Current Opinion in Anaesthesiology, 2016
With recent research trying to explore the pathophysiologic mechanisms behind vasospasm, newer pharmacological and nonpharmacological treatments are being targeted at various pathways involved. This review is aimed at understanding the mechanisms and current and future therapies available to treat vasospasm.Computed tomography perfusion is a useful ...
Ganne S Umamaheswara, Rao +1 more
openaire +2 more sources
With recent research trying to explore the pathophysiologic mechanisms behind vasospasm, newer pharmacological and nonpharmacological treatments are being targeted at various pathways involved. This review is aimed at understanding the mechanisms and current and future therapies available to treat vasospasm.Computed tomography perfusion is a useful ...
Ganne S Umamaheswara, Rao +1 more
openaire +2 more sources
Neurosurgery Clinics of North America, 1998
Established vasospasm is refractory to vasodilators, although certain agents (nimodipine or papavarine) may reverse early vasospasm when administered in high local concentrations. Calcium channel antagonists do not affect the incidence of arteriographic vasospasm and probably improve outcome by other mechanisms.
openaire +2 more sources
Established vasospasm is refractory to vasodilators, although certain agents (nimodipine or papavarine) may reverse early vasospasm when administered in high local concentrations. Calcium channel antagonists do not affect the incidence of arteriographic vasospasm and probably improve outcome by other mechanisms.
openaire +2 more sources
Clinical neurosurgery, 1994
If patients are treated within the first 3 days after rupture, early definitive clipping associated with mechanical clot removal to the extent practical and instillation of fibrinolytic agents seems likely to become standard therapy. Patients should also receive calcium antagonists and be maintained in normal fluid and electrolyte balance. At the first
B, Weir, L, MacDonald
openaire +1 more source
If patients are treated within the first 3 days after rupture, early definitive clipping associated with mechanical clot removal to the extent practical and instillation of fibrinolytic agents seems likely to become standard therapy. Patients should also receive calcium antagonists and be maintained in normal fluid and electrolyte balance. At the first
B, Weir, L, MacDonald
openaire +1 more source