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Hypertriglyceridaemia and Chenodeoxycholic Acid [PDF]
, 1977 Abnormalities of lipoprotein metabolism are frequently found in patient with primary gout. The most common lipid abnormality is hypertriglyceridaemia, independent of, and not secondary to, either obesity, alcohol consumption, or carbohydrate intolerance (1,2).
Alessandro Debolini, Roberto Marcolongo
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Structure of chenodeoxycholic acid in chenodeoxycholic acid ethyl acetate solvate [PDF]
Acta Crystallographica Section C Crystal Structure Communications, 1990 C 24 H 40 O 4 •C 4 H 8 O 2 cristallise dans P6 5 avec a=22,169, c=10,226 A, Z=6; affinement jusqu'a R=0,071. Les molecules acides chenodesoxycholiques sont arrangees en une chaine helicoidale autour de l'axe 6 5 , formant ainsi une colonne infinie. Les groupes hydrophiles externes de ces colonnes sont empiles parallelement l'un a l'autre et connectes
A. Schouten +2 more
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Metabolism of chenodeoxycholic acid in hamsters
Lipids, 1976AbstractThe study on the metabolism after oral administration of chenodeoxycholic acid‐24‐14C was performed by analysis of radioactivity that had appeared in bile and feces of male hamsters. The radioactive bile acids were analyzed by thin layer chromatography and identified by the isotope dilution method. In the bile of the hamsters with bile fistula,
Kouichi Katayama, Tadashi Tateyama
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Solvolysis of chenodeoxycholic acid sulfates
Steroids, 1981Chemical solvolysis of chenodeoxycholic acid sulfates was studied using 4 published methods. Quantitative recovery of chenodeoxycholic acid from the 3-sulfate was obtained with each method. However, only 2 methods yielded chenodeoxycholic acid after solvolysis of the 7-sulfate.
Norman B. Javitt +2 more
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Chenodeoxycholic Acid and Ursodeoxycholic Acid
1990Cholelithiasis (gallstones) is a major medical and economic problem in the USA (Schoenfield, 1977; Schoenfield et al., 1981). It has been estimated to have a prevalence of 15 million women and five million men, and almost one million new cases are discovered each year (Ingelfinger, 1968; Friedman et al., 1966).
Robert A. Maxwell, Shohreh B. Eckhardt
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Toxicity of Chenodeoxycholic Acid in the Rhesus Monkey
Gastroenterology, 1975Chenodeoxycholic acid is an important drug for the treatment of cholesterol cholelithiasis in man. Although no toxicity has been demostrated in man, liver lesions develop in rhesus monkeys treated with chenodeoxycholic acid. To elucidate the mechanism of toxicity, chenodeoxycholic acid.
Thomas Chen +11 more
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The Formation of Deoxycholic Acid and Chenodeoxycholic Acid in Man
Clinical Science, 19741. The turnover of deoxycholic acid and chenodeoxycholic acid was studied in six normolipaemic patients after oral administration of trace amounts of isotopically labelled compounds. 2. The mean values for half-life, pool size and turnover of deoxycholic acid were 3·0 days, 663 mg and 171 mg/day respectively.
Kurt Einarsson, Kjell Hellström
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Biochemical and Biophysical Research Communications, 1975
Abstract Chenodeoxycholic acid (300 mg + 14 C) was administered orally to a bile fistula patient receiving a constant infusion of { 3 H}mevalonic acid. Suppression of endogenous cholic and chenodeoxycholic acid synthesis occurred within 2 to 4 hours and continued for the next 10 hours; synthesis returned to the baseline level after 18 hours ...
L. Gregg Halloran +3 more
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Abstract Chenodeoxycholic acid (300 mg + 14 C) was administered orally to a bile fistula patient receiving a constant infusion of { 3 H}mevalonic acid. Suppression of endogenous cholic and chenodeoxycholic acid synthesis occurred within 2 to 4 hours and continued for the next 10 hours; synthesis returned to the baseline level after 18 hours ...
L. Gregg Halloran +3 more
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Chenodeoxycholic Acid: Uses and Limitations
Hospital Practice, 1979Conclusions about the efficacy and safety of this agent for dissolving cholesterol gallstones must wait until late next year, when results of large clinical trials are available. But results from many studies are available, as are preliminary clinical data from trials of a possible alternative, ursodeoxycholic acid.
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Effect of dietary chenodeoxycholic acid and lithocholic acid in the rabbit [PDF]
The American Journal of Digestive Diseases, 1974 The feeding of chenodeoxycholic acid or lithocholic acid (0.05 or 0.5% of the diet) to rabbits produced cirrhotic and necrotic changes in the liver, accompanied by an increase of secondary bile acids in bile. Animals fed 0.05% lithocholic acid, 0.05% or 0.5% chenodeoxycholic acid, but not those receiving 0.5% lithocholic acid were able to survive for a
E. H. Mosbach +3 more
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