Results 191 to 200 of about 21,815 (286)

Unraveling Inflammatory Pain: A Narrative Review of Molecular Pathways and Multidisciplinary Treatments

open access: yesHealth Science Reports, Volume 9, Issue 6, June 2026.
ABSTRACT Background Inflammatory pain is a widespread global health concern arising from complex molecular pathways triggered by a consequence of tissue damage and immune activation. It significantly impacts quality of life and functional outcomes, necessitating a comprehensive understanding of underlying mechanisms and multidisciplinary approaches for
Fatma Mohamed Elmansy   +6 more
wiley   +1 more source

Relevance of synovial fluid chondroitin sulphate as a biomarker to monitor polo pony joints.

open access: green, 2014
Raquel Yvonne Arantes Baccarin   +3 more
openalex   +1 more source

Subdomain 3 of Plasmodium falciparum VAR2CSA DBL3x Is Identified as a Minimal Chondroitin Sulfate A-binding Region [PDF]

open access: hybrid, 2010
Kavita Singh   +9 more
openalex   +1 more source

Targeting Chondroitin Sulfate Proteoglycans: An Emerging Therapeutic Strategy to Treat CNS Injury [PDF]

open access: bronze, 2020
Nabanita Mukherjee   +9 more
openalex   +1 more source

Ceragenin‐Loaded Tri‐Layered Skin Substitute Composed of Natural and Synthetic Biopolymers for Burn Wound Healing

open access: yesJournal of Biomedical Materials Research Part A, Volume 114, Issue 6, June 2026.
ABSTRACT Burn wounds present significant clinical challenges due to high infection risk, delayed healing, and extensive tissue damage. The development of biomimetic skin substitutes capable of simultaneously supporting tissue regeneration and preventing infection remains a critical need in burn wound management. In this study, a novel ceragenin (CSA‐44)
Nawal Aljayyousi   +9 more
wiley   +1 more source

Reciprocal Macrophage‐MSC Crosstalk Drives Immunomodulatory and Regenerative Phenotypes in a Mineralized Collagen Scaffold

open access: yesJournal of Biomedical Materials Research Part A, Volume 114, Issue 6, June 2026.
Macrophage‐MSC interactions are dynamic. Following injury, neutral (M0) macrophages arrive at the defect site and polarize towards M1 (pro‐inflammatory) macrophages, which can secrete inflammatory and cell recruitment factors. MSCs in this inflammatory defect can take on a licensed state in which they secrete a series of immunomodulatory factors that ...
Vasiliki Kolliopoulos   +7 more
wiley   +1 more source

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