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Lipid-lowering therapy: Evidence and future perspective. [PDF]
JRSM Cardiovasc DisKomiyama M, Wada H, Dan GA, Hasegawa K.
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Addressing chemically-induced obesogenic metabolic disruption: selection of chemicals for in vitro human PPARα, PPARγ transactivation, and adipogenesis test methods. [PDF]
Front Endocrinol (Lausanne)Ozcagli E, Kubickova B, Jacobs MN.
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Straightforward construction of functionalized γ-lactams via conjugated-engineered covalent organic framework photocatalysed cascade reactions. [PDF]
Nat CommunLin X +11 more
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Experimental and In Silico Approaches to Study Carboxylesterase Substrate Specificity. [PDF]
J XenobiotRibone SR, Quevedo MA.
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Integrated bioinformatics and network search identify biomarkers and comorbid mechanisms of atrial fibrillation and ischemic stroke. [PDF]
Medicine (Baltimore)Liu B +9 more
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Clofibrate and Atherosclerosis
New England Journal of Medicine, 1979Despite a variety of efforts aimed at altering its course, atherosclerosis and its sequelae are still major causes of death in the United States and other industrialized nations. In the absence of a single, clearcut etiologic agent, therapeutic interventions have focused on the known multiple risk factors for atherosclerosis, including ...
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2000
Abstract Clofibrate is an oil with a BP of 148°-150°C. It is practi cally insoluble in water but is miscible with ethanol, ace tone, chloroform, and ether (12). Clofibrate and related drugs are branched-chain fatty acid esters. Clofibrate is used in the treatment of hyperlipidemia (types 116, III, IV, and V) and central diabetes ...
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Abstract Clofibrate is an oil with a BP of 148°-150°C. It is practi cally insoluble in water but is miscible with ethanol, ace tone, chloroform, and ether (12). Clofibrate and related drugs are branched-chain fatty acid esters. Clofibrate is used in the treatment of hyperlipidemia (types 116, III, IV, and V) and central diabetes ...
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Probenecid-clofibrate interaction
Clinical Pharmacology and Therapeutics, 1981Clofibric acid disposition was studied in four healthy men after 1 wk of clofibrate ingestion (500 mg orally every 12 hr) with and without probenecid (500 mg orally every 6 hr). Mean (+/- SD) free clofibric acid plasma concentration in the four subjects over a dosage interval at steady state was 2.5 +/- 0.03 mg/1 before and 9.05 +/- 1.09 mg/1 after the
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