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Identification of co-expressed central genes and transcription factors in acute myocardial infarction and diabetic nephropathy. [PDF]
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Clofibrate and Atherosclerosis
New England Journal of Medicine, 1979Despite a variety of efforts aimed at altering its course, atherosclerosis and its sequelae are still major causes of death in the United States and other industrialized nations. In the absence of a single, clearcut etiologic agent, therapeutic interventions have focused on the known multiple risk factors for atherosclerosis, including ...
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Probenecid-clofibrate interaction
Clinical Pharmacology and Therapeutics, 1981Clofibric acid disposition was studied in four healthy men after 1 wk of clofibrate ingestion (500 mg orally every 12 hr) with and without probenecid (500 mg orally every 6 hr). Mean (+/- SD) free clofibric acid plasma concentration in the four subjects over a dosage interval at steady state was 2.5 +/- 0.03 mg/1 before and 9.05 +/- 1.09 mg/1 after the
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