Results 191 to 200 of about 34,207 (226)

Clostridium perfringens type D epsilon intoxication in one‐day‐old calves

Veterinary Record, 2009
Clostridium perfringens type D enterotoxaemia, or ‘pulpy kidney disease’, is a very common cause of sudden death in sheep worldwide. There are numerous scientific publications on the epidemiology and pathology of enterotoxaemia in sheep; in contrast, there have been few studies of type ...
P J, Watson, S F E, Scholes
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Effects of Intravenous Injection of Clostridium perfringens Type D Epsilon Toxin in Calves

Journal of Comparative Pathology, 2002
In cattle, a neurological lesion similar to that produced in sheep and goats by Clostridium perfringens type D enterotoxaemia has been reported. However, no causal relationship has been established between this disease and the lesion in cattle. The effects of single and multiple intravenous injections of epsilon toxin in three calves aged 6 months were
Uzal, F. A., Kelly, W. R., Morris, W. E., Assis, R. A.   +3 more
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AN ENTEROTOXIN PRODUCED BY CLOSTRIDIUM PERFRINGENS TYPE D. PURIFICATION BY AFFINITY CHROMATOGRAPHY

Acta Pathologica Microbiologica Scandinavica Section B Microbiology, 1976
Clostridium perfringens type D 9867 produced enterotoxin immunologically identical to that produced by types A and C which are responsible for C. perfringens food poisoning. Enterotoxin from type C 5386 and type D 9867 was produced in Duncan og Strong sporulation medium (DS medium).
T, Uemura, R, Skjelkvåle
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Vitamin E-Enhanced Humoral Antibody Response to Clostridium Perfringens Type D in Sheep

British Veterinary Journal, 1983
SUMMARY Dietary supplementation of vitamin E ([dl]-α-tocopheryl acetate) significantly increased humoral antibody production in sheep vaccinated with Clostridium perfringens type C and D toxoids, as measured in an ELISA test. A vitamin E adjuvant increased antibody production much more than dietary supplementation.
R P, Tengerdy, D L, Meyer, L H, Lauerman, D C, Lueker, C F, Nockels   +4 more
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A STUDY OF THE SOLUBLE ANTIGENS OF CLOSTRIDIUM PERFRINGENS TYPE D

Canadian Journal of Microbiology, 1964
The soluble antigens of Clostridium perfringens type D have been separated into four fractions by chromatography on DEAE-cellulose. The fractions were examined for their biological activity and for their homogeneity by starch gel electrophoresis, double diffusion in agar, and immunoelectrophoresis.
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Genomic analysis of Clostridium perfringens type D isolates from goat farms

Veterinary Microbiology
C. perfringens type D strains are the leading cause of enterotoxaemia in ruminants such as goats, sheep, and cattle. However, there has been no prior research on the genomic characteristics of C. perfringens type D strains from various regions in China.
Hang, Feng, Ke, Wu, Yuan, Yuan, Mingjin, Fang, Juan, Wang, Ruichao, Li, Rong, Zhang, Xinglong, Wang, Dongyang, Ye, Zengqi, Yang   +9 more
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Antibody Response in Goats Vaccinated with Liposome-adjuvanted Clostridium perfringens Type D Epsilon Toxoid

Veterinary Research Communications, 1999
A trial was performed using 20 goats to evaluate the antibody responses to a liposome-adjuvanted Clostridium perfringens epsilon toxoid vaccine (LIPV). The antibody response was compared with those produced by epsilon toxoid vaccines prepared using aluminium hydroxide (ALV) and incomplete Freud's adjuvant (FAV).
Uzal, F. A., Wong, J. P., Kelly, W. R., Priest, J.   +3 more
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An outbreak of enterotoxaemia caused by Clostridium perfringens type D in goats in Patagonia

Veterinary Record, 1994
Forty-four of a flock of 117 angora goats in the Rio Negro province of Argentina died within four days. Most of the animals died shortly after the onset of clinical signs, but in a few the clinical course lasted for several days. Post mortem the small and large intestines were filled with watery contents, blood and fibrin clots, and there were numerous
F A, Uzal, M I, Pasini, F V, Olaechea, C A, Robles, A, Elizondo   +4 more
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Ultrastructural changes in the brain of mice given Clostridium perfringens type D epsilon toxin

Journal of Comparative Pathology, 1984
Mice were given lethal and sublethal doses of Clostridium perfringens Type D epsilon toxin and the early morphological changes in perfusion-fixed intoxicated brains were examined from 30 min to 6 h post-inoculation. The initial ultrastructural finding was swelling of astrocytes, especially the perivascular extensions of these cells.
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Enterotoxemia (Clostridium perfringens, Type D) Toxoids

Veterinary Clinics of North America: Food Animal Practice, 1990
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