Results 201 to 210 of about 49,823 (238)
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Coagulase-Negative Staphylococci
Southern Medical Journal, 1988Coagulase-negative staphylococci, long considered to be harmless commensals or contaminants, have emerged as major pathogens as medical technology has advanced. They are a major cause of intravenous-catheter-associated bacteremia, endocarditis, otitis media, and infection of joint prostheses, vascular grafts, cardiac pacemakers, cerebrospinal fluid ...
R E, Neihart, J S, Fried, G R, Hodges
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Coagulase-negative Staphylococci
Zentralblatt für Bakteriologie, Mikrobiologie und Hygiene. Series A: Medical Microbiology, Infectious Diseases, Virology, Parasitology, 1987Coagulase-negative staphylococci (CNS) are opportunistic bacteria which undoubtedly are able to cause severe infections in humans and animals. Referring to our own experimental data the actual taxonomic state of staphylococci is reviewed. Furthermore, statements concerning the clinical significance of CNS are given.
G, Pulverer +2 more
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Coagulase-Negative Staphylococci
Infection Control, 1982It is becoming clear that modern medical practices leading to the compromise of patients (e.g., by the ntroduction of prostheses or • -atheters and immunosuppressive therapy) have greatly enhanced the risk of infection by resident coagulase-negative Staphylococcus species.
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COAGULASE-NEGATIVE STAPHYLOCOCCI IN BLOOD-CULTURES
The Lancet, 1959A C, CUNLIFFE, I, JANOTA
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Emergence of coagulase-negative staphylococci
Expert Review of Anti-infective Therapy, 2020Introduction: Compared to Staphylococcus aureus, coagulase-negative staphylococci (CoNS) are characterized by a lower capacity to cause acute, live-threatened infections. CoNS are, however, of ever increasing importance as pathogens causing infections in immunocompromised patients and after foreign-material implantation.
Karsten Becker +4 more
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Glycopeptide Resistance in Coagulase-Negative Staphylococci
European Journal of Clinical Microbiology & Infectious Diseases, 2000Coagulase-negative staphylococci (CNS) were the first organisms in which acquired glycopeptide resistance was recognized. Ever since the early reports, it has been apparent that resistance to teicoplanin is more common than that to vancomycin and that resistance occurs mostly in species such as Staphylococcus haemolyticus and Staphylococcus epidermidis.
F, Biavasco, C, Vignaroli, P E, Varaldo
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Phage typing of coagulase-negative staphylococci
Journal of Clinical Pharmacy and Therapeutics, 1992Seventy-nine staphylococcal strains isolated from blood cultures (57 coagulase-negative staphylococci (CNS) and 22 S. aureus) and 308 CNS isolated from the skin of healthy donors were phage typed. S. epidermidis and S. capitis were readily typed with 91 strains out of 124 and 24 strains out of 43 strains being successful.
Boussard, Paule +3 more
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Coagulase-Negative Staphylococci: Role as Pathogens
Annual Review of Medicine, 1999Coagulase-negative staphylococci have long been regarded as apathogenic but their important role as pathogens and their increasing incidence have been recognized and studied in recent years. Although specific virulence factors are not as clearly established as they are in Staphylococcus aureus, it seems clear that factors such as bacterial ...
Hübner, Johannes, Goldmann, Donald A.
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Endocarditis caused by coagulase-negative staphylococci
Infection, 1979Sixteen patients with coagulase-negative staphylococcal endocarditis were treated at the University of Minnesota Hospitals between January 1970 and September 1977. In six patients, endocarditis developed after prosthetic valve surgery; among the other ten patients (the medical group), eight had known antecedent valvular disease. The skin was thought to
D N, Williams +4 more
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Encapsulation of Coagulase-Negative Staphylococci
Zentralblatt für Bakteriologie, 1989It is becoming clear that encapsulation is frequent among coagulase negative staphylococci and is unrelated to the formation of extracellular polysaccharide slime by many strains. Crude slime may contain capsular polysaccharides or proteins, as well as cell wall components, but this is probably the result of cell wall turnover in growing bacteria.
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