Results 301 to 310 of about 769,092 (333)
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Blood Coagulation

Biochemistry (Moscow), 2002
The process of tissue factor initiated blood coagulation is discussed. Reactions of the blood coagulation cascade are propagated by complex enzymes containing a vitamin K-dependent serine protease and an accessory cofactor protein that are assembled on a membrane surface in a calcium-dependent manner.
S. Butenas, K. G. Mann, null Butenas
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Coagulation Studies

2003
Disseminated intravascular coagulation (DIC) is a complex acquired, coagulopathy resulting from excessive thrombin formation. Abnormal tissue factor (TF) expression is a major mechanism initiating DIC in many disorders, including obstetric complications, sepsis, cancer, and trauma.
Hazelzet, Jan, Hack, CE, Groot, Ronald
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Coagulation Monitoring

Anesthesiology Clinics of North America, 2006
Monitoring hemostasis is now possible by different modalities, of which the point of care devices seem most helpful to the clinician in the operating room. Most of these monitors are being used in the cardiac population, and their significance in other fields remains to be assessed.
Antoine G, Rochon   +1 more
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Blood Coagulation and Coagulation Tests

Medical Clinics of North America, 1984
The hemostatic mechanism has evolved to provide efficient protection from traumatic blood loss and yet maintain the blood in a fluid state in the circulation as a whole. Recent advances in biochemistry have provided both detailed understanding of hemostasis and clinically useful coagulation assays to exploit this understanding.
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Disseminated intravascular coagulation

Seminars in Oncology Nursing, 1999
To provide an overview of the pathophysiology, manifestations, diagnosis, and treatment of disseminated intravascular coagulation (DIC) as it occurs in cancer.Published articles, research reports, and book chapters.The syndrome of DIC is a serious hypercoagulation state that in its acute form may be life-threatening.
Barbara Holmes Gobel, William P. Hogle
  +9 more sources

Coagulation cascade

Hemodialysis International, 2006
AbstractThe coagulation cascade is now recognized to be a series of proteolytic events mainly localized to the surface of activated platelets. Once platelets become activated by exposure to activated endothelium, they release mediators such as P‐selectin and von Willebrand factor that promote microvesicle formation and platelet adherence.
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Coagulation Defects

Anesthesiology Clinics of North America, 2006
The present understanding of the coagulation process emphasizes the final common pathway and the proteolytic systems that result in the degradation of formed clots and the prevention of unwanted clot formations, as well as a variety of defense systems that include tissue repair, autoimmune processes, arteriosclerosis, tumor growth, the spread of ...
Doreen E, Soliman, Lynn M, Broadman
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Cluster Coagulation

Communications in Mathematical Physics, 2000
zbMATH Open Web Interface contents unavailable due to conflicting licenses.
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Blood coagulation

Best Practice & Research Clinical Obstetrics & Gynaecology, 2003
Blood coagulation can be initiated by two pathways: the extrinsic pathway, which is triggered by release of tissue factor from the site of injury, and the intrinsic system, which is stimulated by contact with a negatively charged surface. Following initial triggering, a series of serine proteases are sequentially activated, culminating in the formation
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Blood coagulation

The Lancet, 2000
Under normal circumstances, the coagulation system is balanced in favour of anticoagulation. Thrombin is the key effector enzyme of the clotting cascade. Antagonists of vitamin K inhibit a vitamin-K-dependent post-translational modification of several coagulation proteins, which is required for these proteins to attain a phospholipid-binding ...
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