Results 91 to 100 of about 3,381 (120)

How should cardiac xenotransplantation be initiated in Japan? [PDF]

Surg Today
Saito S, Miyagawa S, Kawamura T, Yoshioka D, Kawamura M, Kawamura A, Misumi Y, Taguchi T, Yamauchi T, Miyagawa S.   +9 more
europepmc   +1 more source

Rare predicted loss-of-function and damaging missense variants in CFHR5 associate with protection from age-related macular degeneration. [PDF]

Am J Hum Genet
Holleman AM, Deaton AM, Hoffing RA, Krohn L, LoGerfo P, Nioi P, Plekan ME, Akle Serrano S, Ticau S, Walshe TE, Borodovsky A, Ward LD.   +11 more
europepmc   +1 more source

The immune system in cardiovascular diseases: from basic mechanisms to therapeutic implications. [PDF]

Signal Transduct Target Ther
Wang X, Chen L, Wei J, Zheng H, Zhou N, Xu X, Deng X, Liu T, Zou Y.   +8 more
europepmc   +1 more source

Limited Proteolysis of Complement Protein C3b by Regulatory Enzyme C3b Inactivator: Isolation and Characterization of a Biologically Active Fragment, C3d,g

Journal of Biochemistry, 1985
Limited proteolysis of C3b by C3b inactivator (factor I) consists of a two-step reaction; rapid cleavage of C3b to yield a nicked C3b derivative, iC3b, and followed by slow cleavage of iC3b to yield two antigenically distinct fragments, C3c and C3d,g.
Tsukasa Seya, Shigeharu Nagasawa, Seya Tsukasa   +2 more
exaly   +4 more sources

Interaction of the fourth component of human complement, treated with amines, chaotropes or frozen-thawed (C4b-like C4), with C4 binding protein and cleavage by C3b/C4b inactivator

Molecular Immunology, 1982
I. von Zabern, Elyse Bloom, Valeria Chu, Irma Gigli   +3 more
openalex   +2 more sources

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On the lysis of paroxysmal nocturnal hemoglobinuria erythrocytes by complement: Dual role of C3b

Blut, 1982
Manfred M Mayer, M M Mayer, Moon L Shin
exaly   +2 more sources

Factor I-dependent inactivation of human complement C4b of the classical pathway by C3b/C4b receptor (CR1, CD35) and membrane cofactor protein (MCP, CD46).

Journal of biochemistry, 1992
Proteolytic inactivation of C4b is a crucial step for regulation of the classical complement pathway. A plasma protease factor I and membrane cofactors, C3b/C4b receptor (CR1) and membrane cofactor protein (MCP), participate in the regulation of cell-bound C4b although the physiological potency of these cofactors remains unknown.
T, Masaki, M, Matsumoto, I, Nakanishi, R, Yasuda, T, Seya   +4 more
openaire   +2 more sources

Interaction of vaccinia virus complement control protein with human complement proteins: Factor I-mediated degradation of C3b to iC3b1 inactivates the alternative complement pathway

Molecular Immunology, 1998
Abstract Vaccinia virus complement control protein (VCP) is a virulence determinant of vaccinia virus that helps protect the virus from the complement attack of the host. To characterize the interaction of VCP with C3 and C4 and understand the mechanism by which VCP inactivates complement, we have expressed VCP in a yeast expression ...
A, Sahu, S N, Isaacs, A M, Soulika, J D, Lambris   +3 more
openaire   +2 more sources

Cyanate as an inactivator of complement proteins.

Journal of Immunology, 1975
Sodium cyanate added to normal human serum or serum from patients with sickle-cell disease resulted in the functional inactivation of C3, C5, C6, C7, and the C3b inactivator, but not C8 and C9.
D. Schultz, P. I. Arnold
semanticscholar   +1 more source

cDNA cloning, sequencing and chromosomal assignment of the gene for mouse complement factor I (C3b/C4b inactivator): identification of a species specific divergent segment in factor I.

Molecular Immunology, 1996
J. Minta, M. Wong, C. Kozak, L. M. Kunnath-Muglia, G. Goldberger   +4 more
semanticscholar   +1 more source