Results 201 to 210 of about 5,539 (231)
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Oral contraception can decrease the pituitary capacity to release gonadotrophins in response to synthetic LH-releasing-hormone

Archiv f�r Gyn�kologie, 1973
The amounts of luteinizing hormone (LH) and follicle stimulating hormone (FSH) released in response to the intravenous administration of 12.5, 25, and 100μg of LH-Releasing Hormone (LH-RH) were found to be severely impeded in 4 out of 5 women who had been taking oral contraceptives for periods ranging from 12 to 36 months.
Robyn, Claude   +4 more
openaire   +2 more sources

The estrogenic activity of synthetic progestins used in oral contraceptives enhances fatty acid synthase-dependent breast cancer cell proliferation and survival

International Journal of Oncology, 2005
Overexpression of the lipogenic enzyme fatty acid synthase (FAS) is a common molecular feature in subsets of sex-steroid-related tumors including breast carcinomas that is associated with poor prognosis. In this study, we explored whether breast-cancer associated FAS (oncogenic antigen-519) is regulated by the progestin component in oral contraceptives.
Javier A, Menendez   +3 more
openaire   +2 more sources

[Synthetic estrogen as a endocrine disruptor--diethylstilbestrol and oral contraceptives].

Nihon rinsho. Japanese journal of clinical medicine, 2001
Recently, considerable attention has been focused on certain environmental contaminants, endocrine disruptors, of industrial origin that may mimic the action of estrogen. Oral contraceptives containing ethynylestradiol, a synthetic estrogen, have been suspected to have adverse environmental effects since prenatal exposure to diethylstilbestrol, another
Y, Takai, O, Tsutsumi
openaire   +1 more source

[Venous and arterial thrombotic events induced by synthetic-steroid hormones or oral contraceptives].

Nihon rinsho. Japanese journal of clinical medicine, 1999
Most users of synthetic-steroid hormones for postmenopausal hormone replacement or contraception, are healthy with a low background incidence of major diseases. Since they take these medicines for a long time, serious or frequent adverse effects should not be occurred.
H, Sugawara, M, Kawakami
openaire   +1 more source

Residual vascular risk of discontinued oral contraception. Role of antibodies to synthetic sex hormones.

Atherosclerosis, 1986
Recent epidemiological data indicate that the risk of thromboembolic disease associated with oral contraception (OC) may persist after discontinuation of the drug. It was demonstrated on the other hand that antibodies to sex steroid hormones which develop in OC users, were significantly correlated with the incidence of thrombosis. It is well known that
V, Beaumont   +3 more
openaire   +1 more source

Synthetic alternatives to Matrigel

Nature Reviews Materials, 2020
Elizabeth A Aisenbrey, William L Murphy
exaly  

Synthetic amorphous silica nanoparticles: toxicity, biomedical and environmental implications

Nature Reviews Materials, 2020
Jonas G Croissant   +2 more
exaly  

Materials design by synthetic biology

Nature Reviews Materials, 2020
Tzu-Chieh Tang   +2 more
exaly  

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