Results 131 to 140 of about 1,861,523 (340)
Mitochondrial transplantation induces macrophage polarization toward an anti‐inflammatory M2 phenotype, enhances their reparative capacities, and facilitates mitochondrial transfer to cardiomyocytes, collectively promoting tissue repair and functional recovery post‐myocardial infarction.
Yuning Zhang+10 more
wiley +1 more source
FURTHER OBSERVATIONS ON THE T WAVE OF THE ELECTROCARDIOGRAM OF THE DOG FOLLOWING THE LIGATION OF THE CORONARY ARTERIES [PDF]
FRED M. SMITH
openalex +1 more source
Coronary Calcification and Coronary Artery Disease
Gottlieb et al. ([1][1]) report computed tomography (CT) calcium scores in 291 patients (73% male) being referred for coronary angiography. The majority (95%) had either intermediate or high pre-test probability of coronary disease, and 42 (19.6%) had presented as emergencies with unstable angina.
openaire +3 more sources
ALKBH5 promoted G3BP1 expression via m⁶A methylation at sites 142/173. G3BP1 interacts with YBX1 and p53, reducing their nuclear translocation and decreasing p53‐mediated SLC7A11 repression. This inhibites cardiomyocyte ferroptosis and mitigates myocardial damage during diabetic ischemia‐reperfusion injury.
Wenyuan Li+5 more
wiley +1 more source
THE CONSTRICTING INFLUENCE OF ADRENALIN UPON THE HUMAN CORONARY ARTERIES [PDF]
Henry G. Barbour
openalex +1 more source
Increased hepatic angiotensinogen (AGT) abundance leads to cardiac diastolic dysfunction via the AngII‐independent pathway. Liver‐derived AGT is internalized by LRP2 in cardiac endothelial cells, subsequently contributing to myocardial diastolic dysfunction by suppressing microvascular angiogenesis via inhibiting the GATA2/Pim3 pathway.
Zetao Heng+7 more
wiley +1 more source
Anatomic Variations in the Coronary Arteries [PDF]
L. Di Guglielmo, M Guttadauro
openalex +1 more source
Atrial Fibroblasts‐Derived Extracellular Vesicles Exacerbate Atrial Arrhythmogenesis
Exosome miR‐224‐5p derived from angiotensin II‐treated atrial fibroblasts creates a substrate for AF by promoting atrial electrical remodeling. Increased exosome miR‐224‐5p enhances AF susceptibility by inhibiting CACNA1c expression and decreasing ICa current of atrial cardiomyocytes.
Yue Yuan+13 more
wiley +1 more source