Results 101 to 110 of about 1,032,826 (402)

Smad7:β-catenin Complex Regulates Myogenic Gene Transcription [PDF]

open access: yes, 2019
Recent reports indicate that Smad7 promotes skeletal muscle differentiation and growth. We previously documented a non-canonical role of nuclear Smad7 during myogenesis, independent of its role in TGF-β signaling.
McDermott, John   +2 more
core   +1 more source

Correlation between Visfatin and Creatine Kinase Levels with Periodontal Health Status of Patients with Coronary Atherosclerosis and Chronic Periodontitis

open access: yesJournal of Baghdad College of Dentistry, 2016
Background: Visfatin is a novel adipokine that mainly secreted by visceral adipose tissue, had an important role in inflammation and immune system. Creatine Kinase (CK) which is an enzyme that is involved in energy metabolism, found in large amounts in ...
Basma R. Omer, Maha Sh .Mahmood
doaj   +1 more source

Creatine kinase monitoring in sport medicine.

open access: yesBritish Medical Bulletin, 2007
UNLABELLED AREAS OF GENERAL AGREEMENT: Total creatine kinase (CK) levels depend on age, gender, race, muscle mass, physical activity and climatic condition.
P. Brancaccio   +2 more
semanticscholar   +1 more source

Results of a nationwide survey of analyses for creatine kinase and creatine kinase isoenzymes.

open access: yesClinical Chemistry, 1984
Abstract More than 300 laboratories participated in an interlaboratory survey of creatine kinase (CK, EC 2.7.3.2) determinations in which they analyzed seven lyophilized samples for total CK and CK isoenzymes and furnished information about their methodology.
T L Hearn   +5 more
openaire   +3 more sources

LncRNA Foxo6os as a Novel “ Scaffold” Mediates MYBPC3 in Combating Pathological Cardiac Hypertrophy and Heart Failure

open access: yesAdvanced Science, EarlyView.
Schematic overview showing that forkhead box O6, opposite strand (Foxo6os) acts as a “scaffold”, directly binding myosin‐binding protein‐C (MYBPC3) and recruiting protein kinase C (PKC‐α) to mediate site‐specific phosphorylation of MYBPC3. This post‐translational modification supports cardiac contraction by regulating L‐type Ca2+ channels, especially ...
Jie Sheng   +9 more
wiley   +1 more source

Serum lipid profiles in acute myocardial infarction patients in Gorgan [PDF]

open access: yes, 2012
Acute myocardial infarction is one of the important reasons of death and unhealthiness in the world. The present study was undertaken to investigate the changes in serum lipids and lipoproteins in patients with acute myocardial infarction. This study was
Marjani, A., Shirafkan, A., Zaker, F.
core  

Comparison of the effects of two types of acute weight loss program (2 and 4%) on the levels of creatine kinase and cardiac troponin enzymes

open access: yesFiyz̤, 2020
Background: One of the most common methods for assessing cardiac damage is the measurement of biological indicators related to heart tissue damage. This study aimed to compare the effects of two types of acute weight loss programs (2 and 4%) on the ...
Farzizade R   +3 more
doaj  

Correlation of anti-signal recognition particle autoantibody levels with creatine kinase activity in patients with necrotizing myopathy.

open access: yesArthritis & Rheumatism, 2011
OBJECTIVE Anti-signal recognition particle (anti-SRP) autoantibodies are associated with severe acquired necrotizing myopathies. The role of these autoantibodies remains elusive, and the evolution of anti-SRP levels over time is unknown.
O. Benveniste   +14 more
semanticscholar   +1 more source

Inhibition of FOXM1 Synergizes with BH3 Mimetics Venetoclax and Sonrotoclax in Killing Multiple Myeloma Cells through Repressing MYC Pathway

open access: yesAdvanced Science, EarlyView.
Relapsed and refractory multiple myeloma remains a major clinical challenge. This study shows that FOXM1 contributes to resistance against BH3 mimetics in multiple myeloma cells. The FOXM1 inhibitor NB73 enhances the effectiveness of BH3 mimetics by reducing FOXM1 expression and suppressing the MYC pathway.
Zhi Wen   +16 more
wiley   +1 more source

Osteopontin ablation ameliorates muscular dystrophy by shifting macrophages to a pro-regenerative phenotype. [PDF]

open access: yes, 2016
In the degenerative disease Duchenne muscular dystrophy, inflammatory cells enter muscles in response to repetitive muscle damage. Immune factors are required for muscle regeneration, but chronic inflammation creates a profibrotic milieu that exacerbates
Barton, Elisabeth R   +7 more
core   +2 more sources

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