Results 71 to 80 of about 2,162,704 (283)

The decay eta_c^prime -> eta_c pi pi

open access: yes, 2002
It is pointed out that the decay of the recently observed charmonium eta_c^prime resonance, eta_c^prime -> eta_c pi pi is simply related to the well studied decay psi^prime -> J/psi pi pi and can thus be used for absolute normalization of other decay ...
Chakravarty S.   +5 more
core   +2 more sources

Molecular dynamics simulations of positively selected codons in FcγRI reveal novel biochemical binding properties

open access: yesFEBS Open Bio, EarlyView.
Evolutionary analysis across 32 placental mammals identified positive selection at residues H148 and W149 in the immune receptor FcγR1. Ancestral reconstruction combined with molecular dynamics simulations reveals how these mutations may influence receptor structure and dynamics, providing insight into the evolution of antibody recognition and immune ...
David A. Young   +7 more
wiley   +1 more source

STALINGRAD: DESTROYED AND REVIVED / СТАЛИНГРАД: РАЗРУШЕННЫЙ И ВОЗРОЖДЕННЫЙ [PDF]

open access: yesСоциология города, 2018
The article deals with the problems of preservation of the architectural objects of Stalingrad after the end of the battle on the Volga river. The authors analyze the results of the examination of buildings in the central part of the city (Ermansky ...
P. P. Oleinikov / П. П. Олейников   +1 more
doaj  

Comparative performance of pxts versus creosote in an awpa e7 field stake test

open access: yesMaderas: Ciencia y Tecnología, 2021
A need exists to develop new organic, environmentally benign wood preservatives for industrial applications to replace the older creosote and pentachlorophenol systems.
Darrel D. Nicholas   +2 more
doaj   +1 more source

Natural Products as Geroprotective Modulators in Diabetic Nephropathy: A Mechanistic Framework Integrating Aging Hallmarks and the AMPK–SIRT1–Nrf2 Axis

open access: yesAging and Cancer, EarlyView.
Natural products target the aging kidney in diabetic nephropathy by restoring the AMPK–SIRT1–Nrf2 axis, reducing oxidative stress, inflammation, fibrosis, and cellular senescence while enhancing mitochondrial biogenesis and antioxidant defenses.
Sherif Hamidu   +8 more
wiley   +1 more source

Decay modes of two repulsively interacting bosons

open access: yes, 2011
We study the decay of two repulsively interacting bosons tunneling through a delta potential barrier by direct numerical solution of the time-dependent Schr\"odinger equation. The solutions are analyzed according to the regions of particle presence: both
Carr L D   +8 more
core   +1 more source

Pion decay

open access: yes
Pedagogical review for a chapter in the Encyclopedia of Particle Physics (published by Elsevier); 25 pages, latex, 11 ...
Bryman, Douglas, Shrock, Robert
openaire   +2 more sources

Predicting Epileptogenic Tubers in Patients With Tuberous Sclerosis Complex Using a Fusion Model Integrating Lesion Network Mapping and Machine Learning

open access: yesAnnals of Clinical and Translational Neurology, EarlyView.
ABSTRACT Objective Accurate localization of epileptogenic tubers (ETs) in patients with tuberous sclerosis complex (TSC) is essential but challenging, as these tubers lack distinct pathological or genetic markers to differentiate them from other cortical tubers.
Tinghong Liu   +11 more
wiley   +1 more source

Trends in Resonance Energy Shifts and Decay Rates for Bose Condensates in a Harmonic Trap

open access: yes, 2001
This is a study of quasi-discrete Bogoliubov quasi-particles in a spherically symmetric harmonic trap. We first evaluate analytically the aymptotic energy shifts of the high energy modes and find them to have $1/sqrt(n)$ dependence on the number of ...
Bergeman, Thomas, Das, Kunal
core   +1 more source

Super‐Refractory Status Epilepticus (SRSE) in a Patient With Compound Heterozygous OPA1 Variants: Case Report and Literature Review

open access: yesAnnals of Clinical and Translational Neurology, EarlyView.
ABSTRACT Objective Super‐Refractory Status Epilepticus (SRSE) is a rare, life‐threatening neurological emergency with unclear etiology in many cases. Mitochondrial dysfunction, often due to disease‐causing genetic variants, is increasingly recognized as a cause, with each gene producing distinct pathophysiological mechanisms.
Pouria Mohammadi   +2 more
wiley   +1 more source

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