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Baillière's Clinical Endocrinology and Metabolism, 1995
The advances in our understanding of the pathophysiology of defects in the antidiuretic hormone, the V2 receptor and the water channel, owing to mutations in the prepro-AVP-NPII, AVPR2 and AQP2 genes respectively, is providing insight into inherited diabetes insipidus as well as the more numerous sporadic cases.
G N, Hendy, D G, Bichet
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The advances in our understanding of the pathophysiology of defects in the antidiuretic hormone, the V2 receptor and the water channel, owing to mutations in the prepro-AVP-NPII, AVPR2 and AQP2 genes respectively, is providing insight into inherited diabetes insipidus as well as the more numerous sporadic cases.
G N, Hendy, D G, Bichet
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Expert Review of Anticancer Therapy, 2006
Water balance is precisely regulated by vasopressin, thirst and the kidneys; plasma osmolality is maintained within a narrow range, despite large variations in normal water intake and loss. Disruption of these finely balanced mechanisms is common, however, and can be precipitated by various disease states.
Shanika, Samarasinghe, Tamara, Vokes
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Water balance is precisely regulated by vasopressin, thirst and the kidneys; plasma osmolality is maintained within a narrow range, despite large variations in normal water intake and loss. Disruption of these finely balanced mechanisms is common, however, and can be precipitated by various disease states.
Shanika, Samarasinghe, Tamara, Vokes
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Medicine, 2005
Diabetes insipidus describes the excess production of dilute urine. It is caused by the lack of production or action of the hormone vasopressin, although primary polydipsia can produce similar symptoms. Diagnosis requires a targeted history and examination.
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Diabetes insipidus describes the excess production of dilute urine. It is caused by the lack of production or action of the hormone vasopressin, although primary polydipsia can produce similar symptoms. Diagnosis requires a targeted history and examination.
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NEPHROGENIC DIABETES INSIPIDUS
Pediatrics, 19551. Two cases of congenital diabetes insipidus resistant to pitressin, or diabetes insipidus of the nephrogenic type, occurring in male cousins during infancy have been described in which the most striking manifestations were recurrent pyrexia, polyuria, polydipsia, poor weight gain and development and hyperelectrolytemia. 2.
J R, WEST, J G, KRAMER
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2018
Diabetes insipidus, characterized by the excretion of copious volumes of unconcentrated urine, results from a deficiency in the action of the antidiuretic hormone arginine vasopressin and can be caused by any of four fundamentally different defects, including impaired secretion (neurohypophyseal diabetes insipidus), impaired renal response (nephrogenic
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Diabetes insipidus, characterized by the excretion of copious volumes of unconcentrated urine, results from a deficiency in the action of the antidiuretic hormone arginine vasopressin and can be caused by any of four fundamentally different defects, including impaired secretion (neurohypophyseal diabetes insipidus), impaired renal response (nephrogenic
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2017
Disruption of water and electrolyte balance is frequently encountered in clinical medicine. Regulating water metabolism is critically important. Diabetes insipidus (DI) presented with excessive water loss from the kidney is a major disorder of water metabolism. To understand the molecular and cellular mechanisms and pathophysiology of DI and rationales
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Disruption of water and electrolyte balance is frequently encountered in clinical medicine. Regulating water metabolism is critically important. Diabetes insipidus (DI) presented with excessive water loss from the kidney is a major disorder of water metabolism. To understand the molecular and cellular mechanisms and pathophysiology of DI and rationales
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Nephrogenic Diabetes Insipidus
Advances in Chronic Kidney Disease, 1998Nephrogenic diabetes insipidus (NDI), which can be inherited or acquired, is characterized by an inability to concentrate urine despite normal or elevated plasma concentrations of the antidiuretic hormone arginine vasopressin (AVP). Polyuria, with hyposthenuria, and polydipsia are the cardinal clinical manifestations of the disease.
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Journal of the American Veterinary Medical Association, 1979
SUMMARY Diabetes insipidus was diagnosed in a 6-year-old Holstein-Friesian cow with a history of recurrent fever, ketosis, lymphadenopathy, and inappetence. The diagnosis was based on the clinical findings, response to exogenous vasopressin, and lack of urine concentration in a water deprivation test.
C E, Wallace, G J, Kociba
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SUMMARY Diabetes insipidus was diagnosed in a 6-year-old Holstein-Friesian cow with a history of recurrent fever, ketosis, lymphadenopathy, and inappetence. The diagnosis was based on the clinical findings, response to exogenous vasopressin, and lack of urine concentration in a water deprivation test.
C E, Wallace, G J, Kociba
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Nephrogenic diabetes insipidus
Current Opinion in Nephrology and Hypertension, 2000Item does not contain ...
Deen, P.M.T. +3 more
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2011
Diabetes insipidus is characterized by polyuria, urine hypotonicity, and hypernatremia. Central diabetes insipidus results from a lack of antidiuretic hormone (ADH); nephrogenic diabetes insipidus results from renal insensitivity to ADH. In the intensive care unit (ICU), diabetes insipidus is most often caused by pituitary surgery, trauma, and brain ...
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Diabetes insipidus is characterized by polyuria, urine hypotonicity, and hypernatremia. Central diabetes insipidus results from a lack of antidiuretic hormone (ADH); nephrogenic diabetes insipidus results from renal insensitivity to ADH. In the intensive care unit (ICU), diabetes insipidus is most often caused by pituitary surgery, trauma, and brain ...
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