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Familial forms of diabetes insipidus: clinical and molecular characteristics
Nature Reviews Endocrinology, 2011 Over the past two decades, the genetic and molecular basis of familial forms of diabetes insipidus has been elucidated. Diabetes insipidus is a clinical syndrome characterized by the excretion of abnormally large volumes of diluted urine (polyuria) and ...
Muriel Babey, Gary Robertson
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Critical Care Medicine, 1992
To review the pathophysiology, diagnosis, and treatment of the syndromes of diabetes insipidus with an emphasis on those situations likely to be encountered in the critical care setting.Extensive clinical experience and relevant publications from the English literature identified via MEDLINE search, citation in reviews, publications of original data ...
L S, Blevins, G S, Wand
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To review the pathophysiology, diagnosis, and treatment of the syndromes of diabetes insipidus with an emphasis on those situations likely to be encountered in the critical care setting.Extensive clinical experience and relevant publications from the English literature identified via MEDLINE search, citation in reviews, publications of original data ...
L S, Blevins, G S, Wand
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Hormone Research in Paediatrics, 2003
Diabetes insipidus is a heterogeneous condition characterized by polyuria and polydipsia caused by a lack of secretion of vasopressin, its physiological suppression following excessive water intake, or kidney resistance to its action. In many patients, it is caused by the destruction or degeneration of the neurons that originate in the supraoptic and ...
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Diabetes insipidus is a heterogeneous condition characterized by polyuria and polydipsia caused by a lack of secretion of vasopressin, its physiological suppression following excessive water intake, or kidney resistance to its action. In many patients, it is caused by the destruction or degeneration of the neurons that originate in the supraoptic and ...
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Baillière's Clinical Endocrinology and Metabolism, 1995
The advances in our understanding of the pathophysiology of defects in the antidiuretic hormone, the V2 receptor and the water channel, owing to mutations in the prepro-AVP-NPII, AVPR2 and AQP2 genes respectively, is providing insight into inherited diabetes insipidus as well as the more numerous sporadic cases.
G N, Hendy, D G, Bichet
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The advances in our understanding of the pathophysiology of defects in the antidiuretic hormone, the V2 receptor and the water channel, owing to mutations in the prepro-AVP-NPII, AVPR2 and AQP2 genes respectively, is providing insight into inherited diabetes insipidus as well as the more numerous sporadic cases.
G N, Hendy, D G, Bichet
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Dimensions of Critical Care Nursing, 2002
The author describes ways to recognize and control this potentially deadly condition.
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The author describes ways to recognize and control this potentially deadly condition.
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Expert Review of Anticancer Therapy, 2006
Water balance is precisely regulated by vasopressin, thirst and the kidneys; plasma osmolality is maintained within a narrow range, despite large variations in normal water intake and loss. Disruption of these finely balanced mechanisms is common, however, and can be precipitated by various disease states.
Shanika, Samarasinghe, Tamara, Vokes
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Water balance is precisely regulated by vasopressin, thirst and the kidneys; plasma osmolality is maintained within a narrow range, despite large variations in normal water intake and loss. Disruption of these finely balanced mechanisms is common, however, and can be precipitated by various disease states.
Shanika, Samarasinghe, Tamara, Vokes
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Aquaporins in Diabetes Insipidus
2023Disruption of water and electrolyte balance is frequently encountered in clinical medicine. Regulating water metabolism is critically important. Diabetes insipidus (DI) presented with excessive water loss from the kidney is a major disorder of water metabolism.
H A Jenny, Lu, Jinzhao, He
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2011
Diabetes insipidus is characterized by polyuria, urine hypotonicity, and hypernatremia. Central diabetes insipidus results from a lack of antidiuretic hormone (ADH); nephrogenic diabetes insipidus results from renal insensitivity to ADH. In the intensive care unit (ICU), diabetes insipidus is most often caused by pituitary surgery, trauma, and brain ...
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Diabetes insipidus is characterized by polyuria, urine hypotonicity, and hypernatremia. Central diabetes insipidus results from a lack of antidiuretic hormone (ADH); nephrogenic diabetes insipidus results from renal insensitivity to ADH. In the intensive care unit (ICU), diabetes insipidus is most often caused by pituitary surgery, trauma, and brain ...
openaire +2 more sources