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Diabetes Insipidus

Dimensions of Critical Care Nursing, 2002
The author describes ways to recognize and control this potentially deadly condition.
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Diabetes insipidus

Baillière's Clinical Endocrinology and Metabolism, 1995
The advances in our understanding of the pathophysiology of defects in the antidiuretic hormone, the V2 receptor and the water channel, owing to mutations in the prepro-AVP-NPII, AVPR2 and AQP2 genes respectively, is providing insight into inherited diabetes insipidus as well as the more numerous sporadic cases.
G N, Hendy, D G, Bichet
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Diabetes insipidus

Expert Review of Anticancer Therapy, 2006
Water balance is precisely regulated by vasopressin, thirst and the kidneys; plasma osmolality is maintained within a narrow range, despite large variations in normal water intake and loss. Disruption of these finely balanced mechanisms is common, however, and can be precipitated by various disease states.
Shanika, Samarasinghe, Tamara, Vokes
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Aquaporins in Diabetes Insipidus

2023
Disruption of water and electrolyte balance is frequently encountered in clinical medicine. Regulating water metabolism is critically important. Diabetes insipidus (DI) presented with excessive water loss from the kidney is a major disorder of water metabolism.
H A Jenny, Lu, Jinzhao, He
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Diabetes Insipidus

Reviews in Endocrine and Metabolic Disorders, 2003
Diabetes insipidus is a heterogeneous condition characterized by polyuria and polydipsia caused by a lack of secretion of vasopressin, its physiological suppression following excessive water intake, or kidney resistance to its action. In many patients, it is caused by the destruction or degeneration of the neurons that originate in the supraoptic and ...
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Diabetes Insipidus

2018
Diabetes insipidus, characterized by the excretion of copious volumes of unconcentrated urine, results from a deficiency in the action of the antidiuretic hormone arginine vasopressin and can be caused by any of four fundamentally different defects, including impaired secretion (neurohypophyseal diabetes insipidus), impaired renal response (nephrogenic
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Nephrogenic diabetes insipidus

Current Opinion in Nephrology and Hypertension, 2000
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Deen, P.M.T.   +3 more
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Diabetes Insipidus

2017
Disruption of water and electrolyte balance is frequently encountered in clinical medicine. Regulating water metabolism is critically important. Diabetes insipidus (DI) presented with excessive water loss from the kidney is a major disorder of water metabolism. To understand the molecular and cellular mechanisms and pathophysiology of DI and rationales
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Nephrogenic Diabetes Insipidus

Annual Review of Physiology, 2001
▪ Abstract  Nephrogenic diabetes insipidus, which can be inherited or acquired, is characterized by an inability to concentrate urine despite normal or elevated plasma concentrations of the antidiuretic hormone arginine vasopressin. Polyuria, with hyposthenuria, and polydipsia are the cardinal clinical manifestations of the disease.
J P, Morello, D G, Bichet
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Nephrogenic Diabetes Insipidus

Advances in Chronic Kidney Disease, 1998
Nephrogenic diabetes insipidus (NDI), which can be inherited or acquired, is characterized by an inability to concentrate urine despite normal or elevated plasma concentrations of the antidiuretic hormone arginine vasopressin (AVP). Polyuria, with hyposthenuria, and polydipsia are the cardinal clinical manifestations of the disease.
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