Results 71 to 80 of about 78,883 (301)

Key messages on sodium and sodium reduction [PDF]

open access: yes
The findings and conclusions in this report are those of the authors and do not necessarily represent the official position of the Centers for Disease Control and Prevention.CS323430Publication date from document properties.sodium-reduction-key-messages ...

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m6A‐Mediated Glycolysis by IL‐37 Drives T Cell Metabolic Reprogramming to Regulate Colitis

open access: yesAdvanced Science, EarlyView.
This study identifies an IL‐37/SIGIRR‐METTL14 regulatory axis that suppresses global m6A modification in CD4+ T cells. IL‐37 signaling, mediated through SIGIRR, inhibits IRAK4 and JNK phosphorylation, leading to downregulation of the methyltransferase METTL14.
Xiaoyan Wang   +26 more
wiley   +1 more source

Sodium Reduction in Communities Program implementation guide [PDF]

open access: yes
The purpose of this guide is to assist food service staf and public health organizations reduce sodium in food service organizations by drawing upon the experiences gained during the Sodium Reduction in Communities Program (SRCP).

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A correlational study of health beliefs and compliance with a sodium-restricted diet [PDF]

open access: yes, 1989
The relationship between health beliefs, based on the Health Beliefs Model, and compliance with a sodium-restricted diet was investigated. Significant predictors of compliance and the relationships among compliance measures were also investigated.
Welch, Mary Ann, 1950-
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A community programme to reduce salt intake and blood pressure in Ghana [PDF]

open access: yes, 2006
Background In Africa hypertension is common and stroke is increasing. Detection, treatment and control of high blood pressure (BP) is limited. BP can be lowered by reducing salt intake.
Kerry, SM   +19 more
core   +1 more source

VDAC1 Upregulation Induces Hyperexcitability of Nociceptive Sensory Neurons Via Enhanced Mitochondrial Atp Efflux in Neuropathic Pain

open access: yesAdvanced Science, EarlyView.
The mechanism diagram of VDAC1 mediating neuronal excitability and neuropathic pain. Briefly, VDAC1 is expressed in DRG neurons and is upregulated following CCI‐induced neuropathic pain. This upregulation enhances ATP transport from mitochondria to the cytoplasm in sensory neurons, leading to increased neuronal excitability and pain behavior.
Fengrun Sun   +7 more
wiley   +1 more source

Food Intake, Diet Quality and Behavioral Problems in Children: Results from the GINI-plus/LISA-plus Studies [PDF]

open access: yes, 2012
Background/Aims: To assess the association between food intake and diet quality and behavioral problems at the 10-year follow-up of the two population-based birth cohorts of the studies German Infant Nutritional Intervention and `Influences of lifestyle ...
Kohlboeck, Gabriele   +36 more
core   +1 more source

Materials‐Guided Gene‐Ionizable Lipid Nanoparticles to Reverse Iron‐Associated Immune Resistance in Renal Cancer

open access: yesAdvanced Science, EarlyView.
ABSTRACT Iron overload is a common metabolic disturbance in cancer and contributes to poor outcomes in renal cell carcinoma (RCC), yet its effects on the tumour immune microenvironment remain unclear. Here we identify a previously unrecognized immunosuppressive axis in which iron overload downregulates the palmitoyltransferase ZDHHC12 in CD8+ T cells ...
Xin Jin   +16 more
wiley   +1 more source

Probing the Honey Bee Diet-Microbiota-Host Axis Using Pollen Restriction and Organic Acid Feeding

open access: yesInsects, 2020
Microbial metabolites are considered important drivers of diet-based microbiota influence on the host, however, mechanistic models are confounded by interactions between diet, microbiota function, and host physiology.
Vincent A. Ricigliano, Kirk E. Anderson
doaj   +1 more source

Sodium Reduction in Communities Program : : outcome evaulation toolkit [PDF]

open access: yes
The purpose of this toolkit is to provide a step-by-step guide for program staff and evaluators who are planning and implementing sodium reduction outcome evaluations.

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