Results 151 to 160 of about 20,405 (323)

Dietary L-3,4-dihydroxyphenylalanine (L-DOPA) augments cuticular melanization in Anopheles mosquitos reducing their lifespan and malaria burden

open access: yesNature Communications
L-3,4-dihydroxyphenylalanine (L-DOPA), a naturally occurring tyrosine derivative, is prevalent in environments that include mosquito habitats, potentially serving as part of their diet.
Emma Camacho   +19 more
doaj   +1 more source

Diagnostic performance of 18F-dihydroxyphenylalanine positron emission tomography in patients with paraganglioma: a meta-analysis

open access: green, 2012
Giorgio Treglia   +7 more
openalex   +2 more sources

THE METABOLISM OF 3,4-DIHYDROXYPHENYLALANINE

open access: yesJournal of Biological Chemistry, 1957
K N, SHAW, A, MCMILLAN, M D, ARMSTRONG
openaire   +2 more sources

A New Phenotype–Genotype Correlation for FIG4 and Parkinson's Disease

open access: yes
Movement Disorders Clinical Practice, EarlyView.
Iro Boura   +4 more
wiley   +1 more source

EFFECT OF EXTRACTION TECHNIQUE OF RUTA GRAVEOLENS ON ANTITYROSINASE ACTIVITY AND CORRELATION AMONGST INHIBITORY ACTIVITY, PHENOLIC COMPOUNDS CONTENT AND CYTOTOXICITY

open access: yesVitae, 2009
Skin pigmentation is the consequence of melanin production and dispersion, this pigment is formed by a successive oxidation of L-Tyrosine into L-Dihydroxyphenylalanine (L-DOPA) and dopaquinone by Tyrosinase enzyme. Skin pigmentation irregularities become
Katalina MUÑOZ D.   +4 more
doaj  

Molecular and cellular determinants of L-Dopa-induced dyskinesia in Parkinson’s Disease

open access: yesnpj Parkinson's Disease
Treatment with L-3,4-dihydroxyphenylalanine (L-Dopa) compensates for decreased striatal dopamine (DA) levels and reduces Parkinson’s disease (PD) symptoms.
Federica Servillo   +22 more
doaj   +1 more source

The Second Hit Hypothesis in Animal and Human Dystonia: The Role of Peripheral Nerve Trauma and Spinal Cord Injury

open access: yesMovement Disorders, EarlyView.
The “second‐hit” hypothesis proposes that both a genetic predisposition and an environmental insult—such as peripheral nerve trauma or spinal cord injury—are required for dystonia development. This review explores how neuroinflammation and maladaptive plasticity, triggered by nerve and spinal cord injury, contribute to dystonia pathogenesis.
Lisa Harder‐Rauschenberger   +1 more
wiley   +1 more source

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