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Amyloid-β and Tau in Alzheimer's disease: pathogenesis, mechanisms, and interplay. [PDF]
Cell Death DisAbdulkhaliq AA +9 more
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Co- and multipathologies need serious consideration for understanding Parkinson's disease pathogenesis and designing novel experimental therapeutic strategies. [PDF]
J Parkinsons DisBoehringer A +3 more
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Castleman Disease Pathogenesis
Hematology/Oncology Clinics of North America, 2018Castleman disease (CD) describes a group of heterogeneous disorders with common lymph node histopathologic features, including atrophic or hyperplastic germinal centers, prominent follicular dendritic cells, hypervascularization, polyclonal lymphoproliferation, and/or polytypic plasmacytosis.
David C, Fajgenbaum, Dustin, Shilling
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THE PATHOGENESIS OF GRAVES' DISEASE
Endocrinology and Metabolism Clinics of North America, 1998Graves' disease, one of the autoimmune thyroid diseases, is caused by the production of IgG autoantibodies directed against the thyrotropin receptor. These antibodies bind to and activate the receptor, causing the autonomous production of thyroid hormones.
B, McIver, J C, Morris
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Pathogenesis of Parkinson’s Disease
Neurologic ClinicsBoth genetic and environmental factors modulate the risk of Parkinson's disease. In this article, all these pathophysiologic processes that contribute to damages at the tissue, cellular, organelle, and molecular levels, and their effects are talked about.
Bin, Xiao +3 more
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The Pathogenesis of Mucosal Disease
Veterinary Clinics of North America: Food Animal Practice, 1995The pathogenesis of MD is complex and remains somewhat obscure. Clearly, the disease occurs in cattle persistently infected with noncytopathic BVDV. It also is clear that cytopathic BVDV is associated with MD, and is the likely trigger of the cellular destruction that leads to clinical disease.
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The Pathogenesis of Graves’ Disease
Endocrine Practice, 1995We have hypothesized over many years that Graves' disease (GD) and the other autoimmune thyroid diseases (AITD) are each due to antigen-specific defects in suppressor (regulatory) T lymphocyte function. There have been several reports dealing with the role of regulatory T lymphocyte subsets, ie., that will prevent autoimmune disease in these and other ...
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The pathogenesis of Hirschsprung disease
Current Opinion in Gastroenterology, 2002Hirschsprung disease is the most common congenital malformation of the enteric nervous system. Phenotypic expression is variable because of incomplete penetrance, and the pathogenesis is multifactorial. Although mutations of the RET tyrosine kinase gene remain the most commonly identified cause, there are now eight separate human gene loci identified ...
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Pathogenesis of Wilson disease
2017Wilson disease is an autosomal-recessive disorder originating from a genetic defect in the copper-transporting ATPase ATP7B that is required for biliary copper secretion and loading of ceruloplasmin with copper. Impaired ATP7B function in Wilson disease results in excessive accumulation of copper in liver, brain, and other tissues.
Ivo Florin, Scheiber +2 more
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