Results 161 to 170 of about 1,587,717 (295)
Expanding disease resistance: engineered NLRS for broad-spectrum protection. [PDF]
Zhou T, Wang J, Wang Y.
europepmc +1 more source
Iron deficiency anemia of unknown etiology and/or resistance to the treatment: The sole manifestation of adult celiac disease (CD) [PDF]
Mustafa Nuri Yenerel +1 more
openalex +1 more source
The present invention relates to methods for producing plants having enhanced disease resistance. NRCl proteins and nucleic acid sequences encoding these are provided, as well as transgenic plants producing NRCl proteins.
Gabriëls, S.H.E.J. +3 more
openaire +1 more source
We show that the majority of the 18 analyzed recurrent cancer‐associated ERBB4 mutations are transforming. The most potent mutations are activating, co‐operate with other ERBB receptors, and are sensitive to pan‐ERBB inhibitors. Activating ERBB4 mutations also promote therapy resistance in EGFR‐mutant lung cancer.
Veera K. Ojala +15 more
wiley +1 more source
Targeted expansion of a barley genebank core collection facilitates the discovery of disease resistance loci. [PDF]
Yuan Z +10 more
europepmc +1 more source
Peroxidasin enables melanoma immune escape by inhibiting natural killer cell cytotoxicity
Peroxidasin (PXDN) is secreted by melanoma cells and binds the NK cell receptor NKG2D, thereby suppressing NK cell activation and cytotoxicity. PXDN depletion restores NKG2D signaling and enables effective NK cell–mediated melanoma killing. These findings identify PXDN as a previously unrecognized immune evasion factor and a potential target to improve
Hsu‐Min Sung +17 more
wiley +1 more source
Effects of soybean intercropping density on photosynthetic characteristics and disease resistance in tobacco. [PDF]
Liu X +12 more
europepmc +1 more source
Active defense as a mechanism of resistance in pine against pine wilt disease.
Hachiro Oku +2 more
openalex +2 more sources
Single circulating tumor cells (sCTCs) from high‐grade serous ovarian cancer patients were enriched, imaged, and genomically profiled using WGA and NGS at different time points during treatment. sCTCs revealed enrichment of alterations in Chromosomes 2, 7, and 12 as well as persistent or emerging oncogenic CNAs, supporting sCTC identity.
Carolin Salmon +9 more
wiley +1 more source

