Results 231 to 240 of about 16,889 (275)

Drug-Induced and Other Orofacial-Cervical Dyskinesias

Annals of Internal Medicine, 1981
Tardive dyskinesia is one of the most prevalent and disabling of the iatrogenic disorders. Characterized by involuntary movements of the orofacial-cervical musculature that develop after prolonged use of neuroleptic drugs, it is sometimes confused with other involuntary movements involving predominantly the head and neck region.
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Neuroleptic drug-induced extrapyramidal syndromes and tardive dyskinesia

Schizophrenia Research, 1991
Neuroleptic (antipsychotic) drug-induced acute extrapyramidal syndromes (EPS) and the late onset tardive dyskinesia (TD) are the major side effects that limit the use of these highly efficacious agents. The appropriate strategy for controlling these side effects is based on the clinical presentations, pathophysiological mechanisms, and contributions of
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Underrecognition of tardive dyskinesia and drug-induced parkinsonism by psychiatric residents

General Hospital Psychiatry, 1992
Recognition of tardive dyskinesia (TD) and other neuroleptic, drug-induced, extrapyramidal side effects presents a major challenge in modern clinical psychopharmacology. Failure to recognize these disorders can lead to poor patient care and may contribute to societal pressure for external control of psychiatric practice.
T E, Hansen, W L, Brown, R M, Weigel, D E, Casey   +3 more
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Neuroradiological Covariates of Drug-Induced Parkinsonism and Tardive Dyskinesia in Schizophrenia

International Journal of Neuroscience, 1991
Computed tomographic (CT) studies have demonstrated structural brain abnormalities including cortical atrophy and enlarged lateral ventricles in a subset of schizophrenic patients including those with abnormal involuntary movements. In the following series of studies, we present our findings pertaining to neuroradiological covariates of drug-induced ...
R, Sandyk, S R, Kay
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Tardive dyskinesia, mild drug-induced dyskinesia, and drug-induced parkinsonism: risk factors and topographic distribution.

Acta neurologica Belgica, 1999
This study attempted to contribute to the subtyping of tardive dyskinesia (TD) by studying the effects of age, sex, psychiatric diagnosis and duration of illness on the severity and topographic distribution of dyskinesia, and to describe the topographic distribution of drug-induced dyskinesia (DID) and drug-induced parkinsonism (DIP) in detail by ...
M C, Akbostanci, E C, Atbaşoğlu, H, Balaban   +2 more
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TREATMENT OF DRUG-INDUCED DYSKINESIAS

The Lancet, 1970
A, Villeneuve, Z, Böszörményi
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Tryptophan in the amelioration of drug-induced tardive dyskinesia

1990
The administration of 8 g L-tryptophan/day to an insomniac patient, suffering from tardive dyskinesia (TD), to promote sleep led to the discovery of an amelioration of the TD symptoms [1].
H. Fisher, G. C. Wagner, L. Kozell, S. K. Johnson, R. Sandyk   +4 more
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Drug-induced dyskinesia in monkeys.

Advances in neurology, 1975
Easily definable dyskinetic syndromes have been produced by injection of L-DOPA, apomorphine, trivastal, carbachol, sodium azide, tetrahydrocannabinol, phenothiazines, and amphetamines into one or more sites. Three species of monkeys have been used: rhesus, green, and squirrel. The descriptive information available on each of these dyskinetic syndromes
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Drug induced dyskinesia: reality or myth?

Diseases of the nervous system, 1975
Psychotropic medication employed in the long-term therapy of mental patients is etiologically linked to oral dyskinesia. Although the term "oral dyskinesia" is used interchangeably with "drug-induced dyskinesia" this author doesn't find sufficient scientific evidence from published studies to implicate the psychotropic drugs solely responsible for ...
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Drug-Induced Dyskinesias

AJN, American Journal of Nursing, 1982
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