Results 121 to 130 of about 23,636 (243)

Identification of a chromosome 6-encoded dystrophin-related protein.

open access: hybrid, 1990
T.S. Khurana   +2 more
openalex   +1 more source

N‐terminal domain of dystrophin [PDF]

open access: bronze, 1994
Armelle Bonet‐Kerrache   +2 more
openalex   +1 more source

Tissue distribution of the dystrophin-related gene product and expression in the mdx and dy mouse.

open access: green, 1991
Donald R. Love   +9 more
openalex   +1 more source

Heterogeneity of the 59-kDa dystrophin-associated protein revealed by cDNA cloning and expression.

open access: hybrid, 1994
Baoli Yang   +4 more
openalex   +1 more source

Inhibition of mTOR attenuates the initiation and progression of BRCA1‐associated mammary tumors

open access: yes
Cancer Communications, Volume 45, Issue 4, Page 486-490, April 2025.
Hye Jung Baek   +13 more
wiley   +1 more source

DNA hypermethylation preceded by H3K27 trimethylation is linked to downregulation of gene expression in disuse muscle atrophy in male mice

open access: yesPhysiological Reports, Volume 13, Issue 7, April 2025.
Abstract Disuse muscle atrophy can result in downregulated gene expression vital to muscle integrity, yet the mechanisms driving this downregulation remain unclear. Epigenetic alterations regulate transcriptional potential, with repressive changes suppressing gene expression.
Junya Shimizu, Fuminori Kawano
wiley   +1 more source

QUANTITATIVE ANALYSIS OF DYSTROPHIN IN HUMAN AND RODENT MUSCLES

open access: bronze, 1992
Ritsuko Koga   +5 more
openalex   +2 more sources

A semi‐automated observation approach to quantify mouse skeletal muscle differentiation using immunohistochemistry

open access: yesPhysiological Reports, Volume 13, Issue 7, April 2025.
In this study, we developed a semi‐automated method to quantify Pax‐7 positive cells in skeletal muscle. The results of the developed semi‐automated method showed good correlation with the manual method, and the analysis time was reduced to one‐twentieth of the manual method.
Kenta Shimizu   +8 more
wiley   +1 more source

Shorter Phosphorodiamidate Morpholino Splice-Switching Oligonucleotides May Increase Exon-Skipping Efficacy in DMD

open access: yesMolecular Therapy: Nucleic Acids, 2018
Duchenne muscular dystrophy is a fatal muscle disease, caused by mutations in DMD, leading to loss of dystrophin expression. Phosphorodiamidate morpholino splice-switching oligonucleotides (PMO-SSOs) have been used to elicit the restoration of a ...
Ugur Akpulat   +6 more
doaj  

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