Results 171 to 180 of about 34,637 (221)

Excess dietary potassium raises blood pressure in male mice by an aldosterone-dependent increase in ENaC. [PDF]

Commun Biol
Assmus AM, Odgaard LN, Garrelds IM, Danser AHJ, Matchkov V, Welling PA, Fenton RA.   +6 more
europepmc   +1 more source

Aldosterone: From Essential Tubular Regulator to Pathological Driver-Physiology, Disease, and Therapeutic Advances. [PDF]

Int J Mol Sci
Strizzi CT, D'Ambrosio V, Grandaliano G, Pesce F.   +3 more
europepmc   +1 more source

Hyperconcentrated mucus in small airways: a mechanistic model for the pathogenesis of paediatric bronchiectasis. [PDF]

Eur Respir Rev
Chatziparasidis G, Chang AB, Bush A, Kantar A, Priftis KN.   +4 more
europepmc   +1 more source

Saliva-driven surface-engineered <i>Bacteroides thetaiotaomicron</i> alleviates hypertension. [PDF]

Bioact Mater
Xu S, Luo H, Du LJ, Dong T, Zhou LJ, Chen BY, Li YL, Tian GC, Meng XQ, Bai XB, Ye HL, Zhang J, Lin WZ, Zhang WC, Liu J, Duan SZ.   +15 more
europepmc   +1 more source

Transgenically expressed human delta epithelial sodium channel facilitated fluid absorption in mouse fetal lung explants. [PDF]

Am J Physiol Lung Cell Mol Physiol
Jain KG, Zhao R, Zhang J, Ji HL.
europepmc   +1 more source

HD-6mAPred: a hybrid deep learning approach for accurate prediction of N6-methyladenine sites in plant species. [PDF]

PeerJ
Li H, Gao W, Tang Y, Guo X.
europepmc   +1 more source

Nedd4-2 ablation in kidney improves glycaemic control in diabetic mice. [PDF]

Cell Death Dis
Manning JA, Jesudason S, Moretti PAB, Pitson SM, Chou ASY, Shabbar M, Saad S, Pollock C, Kumar S.   +8 more
europepmc   +1 more source

Regulating ENaC’s gate

American Journal of Physiology - Cell Physiology, 2020
Epithelial Na+ channels (ENaCs) are members of a family of cation channels that function as sensors of the extracellular environment. ENaCs are activated by specific proteases in the biosynthetic pathway and at the cell surface and remove embedded inhibitory tracts, which allows channels to transition to higher open-probability states.
Thomas R Kleyman, Douglas C Eaton
exaly   +4 more sources

Some of the next articles are maybe not open access.

Related searches:

Peptide Inhibition of ENaC

Biochemistry, 1998
Liddle's disease is an autosomal dominant form of human hypertension resulting from a basal activation of amiloride-sensitive Na+ channels (ENaC). This channel activation is produced by mutations in the beta- and/or gamma-carboxy-terminal cytoplasmic tails, in many cases causing a truncation of the last 45-76 amino acids.
Ismailov, I I, Shlyonsky, Vadim, Serpersu, E H, Fuller, C M, Cheung, H C, Muccio, D, Berdiev, B K, Benos, D J   +7 more
openaire   +2 more sources