Results 71 to 80 of about 100,569 (330)
ENO Schemes with Subcell Resolution* [PDF]
In this paper, we introduce the notion of subcell resolution, which is based on the observation that unlike point values, cell-averages of a discontinuous piecewise-smooth function contain information about the exact location of the discontinuity within the cell.
openaire +2 more sources
Gene transfer of endothelial nitric oxide synthase (eNOS) in eNOS-deficient mice [PDF]
Relaxation to acetylcholine (ACh) and calcium ionophore (A-23187) is absent in aortas from endothelial nitric oxide synthase (eNOS)-deficient (eNOS -/-) mice. We hypothesized that gene transfer of eNOS would restore relaxation to ACh and A-23187 in eNOS -/- mice.
Kristy D. Lake-Bruse+5 more
openaire +3 more sources
Therapies that raise levels of HDL, which is thought to exert atheroprotective effects via effects on endothelium, are being examined for the treatment or prevention of coronary artery disease (CAD).
C. Besler+25 more
semanticscholar +1 more source
Amphibious Generator via Mechanical Coupling for Versatile Energy Harvesting
An amphibious hybrid triboelectric nanogenerator–electromagnetic generator (TENG–EMG) efficiently harvests energy from wind and water. By integrating TENG and EMG units with a gear‐assisted mechanism, it enhances power output and powers small electronic devices.
Chi Zhang+7 more
wiley +1 more source
eNOS uncoupling in pulmonary hypertension [PDF]
This editorial refers to ‘Pulmonary hypertension in adult Alk1 heterozygous mice due to oxidative stress’ by M. Jerkic et al. , pp. 375–384, this issue. Pulmonary arterial hypertension (PAH) is characterized by high blood pressure and vascular remodelling in pulmonary arteries and subsequently right ventricular hypertrophy.
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Traditional static culture provides limited physiological relevance for evaluation of vascular grafts. A perfusion bioreactor system can provide more accurate in vitro model for evaluation of vascular grafts, exposing endothelial cells on 3D structure to physiological or pathological shear stress.
Praveesuda L. Michael+10 more
wiley +1 more source
Background Glutathionylation of endothelial nitric oxide synthase (eNOS) “uncouples” the enzyme, switching its function from nitric oxide (NO) to O2•− generation.
K. Galougahi+10 more
semanticscholar +1 more source
Bathochromic shift‐type OHC (b‐OHC) was found in donor–acceptor (D–A) type pyridinium dye bearing halide counter anion, as well as donor‐π‐acceptor (D‐π‐A) type pyridinium dye. This work revealed that formation of halogen bond (XB) or complex such as [R─X·Y]− between halogen atom (X) of organohalogen molecule and counter anion (Y−) of dye molecule ...
Kumpei Kozuka+2 more
wiley +1 more source
Aim. To study the effects of eNOS gene polymorphisms (Glu298Asp and VNTR intron 4) on endothelial dysfunction (ED) development, chronic heart failure (CHF) severity and progression in patients with coronary heart disease (CHD) and arterial hypertension ...
N. F. Yakovleva+7 more
doaj
Role of eNOS phosphorylation at Ser-116 in regulation of eNOS activity in endothelial cells [PDF]
Endothelial nitric oxide synthase (eNOS) catalyzes the conversion of L-arginine to L-citrulline and nitric oxide (NO), an important modulator of vascular function. eNOS is regulated post-translationally through phosphorylation/dephosphorylation at a number of specific phosphorylation sites including Ser-116 in the bovine eNOS sequence.
Richard C. Venema+5 more
openaire +3 more sources