Results 11 to 20 of about 6,639 (199)

Sample processing for DNA chip array-based analysis of enterohemorrhagic Escherichia coli (EHEC) [PDF]

open access: yesMicrobial Cell Factories, 2008
Abstract Background Exploitation of DNA-based analyses of microbial pathogens, and especially simultaneous typing of several virulence-related genes in bacteria is becoming an important objective of public health these days.
Basselet, Pascal   +3 more
openaire   +6 more sources

Introduction to the Special Issue “Molecular Basis and the Pathogenesis of Enterohemorrhagic Escherichia coli Infections”

open access: yesToxins, 2020
Although much of the world has progressed since the 1980s, our ability to treat infections with enterohemorrhagic Escherichia coli (EHEC) has unfortunately shown little improvement [...]
Kim Stanford
doaj   +2 more sources

Analysis of Collection of Hemolytic Uremic Syndrome–associated Enterohemorrhagic Escherichia coli [PDF]

open access: yesEmerging Infectious Diseases, 2008
Multilocus sequence typing of 169 non-O157 enterohemorrhagic Escherichia coli (EHEC) isolated from patients with hemolytic uremic syndrome (HUS) demonstrated 29 different sequence types (STs); 78.1% of these strains clustered in 5 STs.
Alexander Mellmann   +8 more
doaj   +2 more sources

TccP2 of O157:H7 and Non-O157 Enterohemorrhagic Escherichia coli (EHEC): Challenging the Dogma of EHEC-Induced Actin Polymerization [PDF]

open access: yesInfection and Immunity, 2007
ABSTRACT Enterohemorrhagic Escherichia coli (EHEC) O157:H7 and enteropathogenic E. coli (EPEC) trigger actin polymerization at the site of bacterial adhesion by inducing different signaling pathways. Actin assembly by EPEC requires tyrosine phosphorylation of Tir, which subsequently
Yoshitoshi, Ogura   +16 more
openaire   +4 more sources

Discrimination of Enterohemorrhagic Escherichia coli (EHEC) from Non-EHEC Strains Based on Detection of Various Combinations of Type III Effector Genes [PDF]

open access: yesJournal of Clinical Microbiology, 2013
ABSTRACT Enterohemorrhagic Escherichia coli (EHEC) strains comprise a subgroup of Shiga-toxin (Stx)-producing E. coli (STEC) and are characterized by a few serotypes. Among these, seven priority STEC serotypes (O26:H11, O45:H2, O103:H2, O111:H8, O121:H19, O145:H28, and O157:H7) are ...
Sabine, Delannoy   +2 more
openaire   +3 more sources

Glycan-Adhering Lectins and Experimental Evaluation of a Lectin FimH Inhibitor in Enterohemorrhagic Escherichia coli (EHEC) O157:H7 Strain EDL933. [PDF]

open access: yesInt J Mol Sci, 2022
In this study, we tried to develop a FimH inhibitor that inhibits adhesion of enterohemorrhagic Escherichia coli (EHEC) on the epithelium of human intestine during the initial stage of infections.
Park JY, Kim CH, Cho SH.
europepmc   +2 more sources

Fitness of Enterohemorrhagic Escherichia coli (EHEC)/Enteroaggregative E. coli O104:H4 in Comparison to That of EHEC O157: Survival Studies in Food and In Vitro. [PDF]

open access: yesAppl Environ Microbiol, 2016
ABSTRACT In 2011, one of the world's largest outbreaks of hemolytic-uremic syndrome (HUS) occurred, caused by a rare Escherichia coli serotype, O104:H4, that shared the virulence profiles of Shiga toxin-producing E. coli (STEC)/enterohemorrhagic E. coli (EHEC)
Böhnlein C   +4 more
europepmc   +4 more sources

Enterohemorrhagic E. coli (EHEC) pathogenesis

open access: yesFrontiers in Cellular and Infection Microbiology, 2012
Enterohemorrhagic Escherichia coli (EHEC) serotype O157:H7 is a human pathogen responsible for outbreaks of bloody diarrhea and hemolytic uremic syndrome worldwide.
Y Nguyen   +2 more
exaly   +3 more sources

Ribosome State Distributions Define Escherichia coli Persister Physiology: Links to Formation, Stress Responses, and Resuscitation Dynamics. [PDF]

open access: yesMicrob Biotechnol
The AUCS70/AUCS100 ratio, derived from ribosome sedimentation profiling, quantitatively distinguishes Escherichia coli persisters from growing cells across multiple stress conditions and identifies RMF as the primary target for anti‐persistence strategies. ABSTRACT Persister cells survive antibiotic exposure and contribute to infection relapse, yet the
Kim H, Song S.
europepmc   +2 more sources

Type VI secretion system degeneration accelerates intestinal epithelial cell death in Escherichia coli O157:H7. [PDF]

open access: yesPLoS Pathogens
The type VI secretion system (T6SS) is a specific protein secretion apparatus that contributes to bacterial virulence. Enterohemorrhagic Escherichia coli O157:H7 (EHEC) harbors multiple prophages and can cause severe human diseases worldwide.
Zhibin Sun   +6 more
doaj   +2 more sources

Home - About - Disclaimer - Privacy