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An anti-estradiol effect of ?-estradiol-17β (?-estradiol)
Steroids, 1969Abstract The 1 -enantiomorph of estradiol-17β will inhibit the effects of estradiol-17β on uterine growth and vaginal opening in immature female mice and on the vaginal smears of spayed adult mice. The inhibitory ratios of ent - to natural estradiol are similar to ratios shown by Terenius to inhibit the incorporation of radio-active ...
Richard A. Edgren, Robert C. Jones
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Estradiol and chlordecone interactions with the estradiol receptor
Toxicology and Applied Pharmacology, 1989The in vivo and in vitro effects of the chlorinated pesticide, chlordecone, on the estradiol receptor of adult ovariectomized CDF-344 rats were examined. Chlordecone's competition with [3H]estradiol for binding to the estradiol receptor in vitro was similar whether receptors were derived from neural or uterine tissue.
Kris Eckols +2 more
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Synthesis of [2‐d]estradiol, [4‐d]estradiol, [2‐t]estradiol and [4‐t]estradiol with high specificity
Journal of Labelled Compounds and Radiopharmaceuticals, 1984Abstract17‐β‐Estradiol has been labeled in the 2 or 4 position with tritium or deuterium. Both 2‐ and 4‐iodoestradiol were prepared by treatment of estradiol with N‐iodosuccinimide in ethanol followed by chromatographic separation. Reductive dehalogenation of 2‐ and 4‐iodoestradiol with tritium gas gave [2‐t] estradiol and [4‐t] estradiol, respectively,
Mont R. Juchau +3 more
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Best Practice & Research Clinical Endocrinology & Metabolism, 2016
Ovarian steroids, such as estradiol (E2), control a vastness of physiological processes, such as puberty, reproduction, growth, development and metabolic rate. In fact, physiological, pathological, pharmacological or genetically-induced estrogen deficiency causes increased appetite and reduced energy expenditure, promoting weight gain and ultimately ...
Manuel Tena-Sempere +2 more
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Ovarian steroids, such as estradiol (E2), control a vastness of physiological processes, such as puberty, reproduction, growth, development and metabolic rate. In fact, physiological, pathological, pharmacological or genetically-induced estrogen deficiency causes increased appetite and reduced energy expenditure, promoting weight gain and ultimately ...
Manuel Tena-Sempere +2 more
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The Effect of Mercury Estradiol on 17β-Estradiol Dehydrogenase
Gynecologic and Obstetric Investigation, 1970The effects of two organic mercurials (4-acetatomercuri-17Β-estradiol and <i>p</i>-chloromercuribenzoate) on human placental estradiol dehydrogenase were evaluated by equilibrium rate exchange and initial velocity measurements. The steroid mercurial was more effective in reducing isotopic exchange between steroid pairs than between
G. Betz, P. Susee
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Endocrinology, 1974
Groups of female rats were decapitated at 3-hr intervals throughout 4-day estrous cycles and the plasma of each rat was assayed for LH, FSH, prolactin, progesterone and estradiol-17β.
R. Butcher, W. E. Collins, N. W. Fugo
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Groups of female rats were decapitated at 3-hr intervals throughout 4-day estrous cycles and the plasma of each rat was assayed for LH, FSH, prolactin, progesterone and estradiol-17β.
R. Butcher, W. E. Collins, N. W. Fugo
semanticscholar +1 more source
An Alternative Route to 2-Bromo-Estradiols and 2-Iodo-Estradiols From Estradiol
Australian Journal of Chemistry, 1987Through careful choice of reaction conditions, alkylation of estradiol with chloromethyl methyl ether may yield either the 3,17β-bis(methoxymethy1) ether or the 3-methoxymethyl ether derivative. Treatment of either of these protected estradiols with s- butyllithium, then with trimethylsilyl chloride affords, regioselectively, the 2-trimethylsilyl
DD Ridley, DJ Pert
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