Results 251 to 260 of about 1,472,047 (314)
Some of the next articles are maybe not open access.
Selected opioids, ethanol and intake of ethanol
Alcohol, 1984Rats were given an opportunity to drink tap water or a sweetened ethanol solution once a day. Across initial days of opportunity, rats increased their intake of the ethanol solution. Prior to some days' sessions with presented fluids, rats received either an injection of placebo (the carrier of drugs) or doses of ethylketocyclozocine, diprenorphine, or
G A, Hunter +3 more
openaire +2 more sources
Alcoholism: Clinical and Experimental Research, 1984
The interaction of ethanol with lipid metabolism is complex. When ethanol is present, it becomes a preferred fuel for the liver and displaces fat as a source of energy. This favors fat accumulation. In addition, the altered redox state secondary to the oxidation of ethanol promotes lipogenesis, for instance, through enhanced formation of acylglycerols.
C S, Lieber, M, Savolainen
openaire +2 more sources
The interaction of ethanol with lipid metabolism is complex. When ethanol is present, it becomes a preferred fuel for the liver and displaces fat as a source of energy. This favors fat accumulation. In addition, the altered redox state secondary to the oxidation of ethanol promotes lipogenesis, for instance, through enhanced formation of acylglycerols.
C S, Lieber, M, Savolainen
openaire +2 more sources
Annals of Emergency Medicine, 1991
Ethanol sensitivity is a syndrome of flushing, tachycardia, weakness, fatigue, and other dysphoric symptoms in response to relatively small doses of ethanol. We describe a case of extreme ethanol sensitivity presenting with coma and review the pathophysiology of the syndrome.
B, Lerman, R, Bodony
openaire +2 more sources
Ethanol sensitivity is a syndrome of flushing, tachycardia, weakness, fatigue, and other dysphoric symptoms in response to relatively small doses of ethanol. We describe a case of extreme ethanol sensitivity presenting with coma and review the pathophysiology of the syndrome.
B, Lerman, R, Bodony
openaire +2 more sources
Acta Neurologica Scandinavica, 2009
Two groups of alcoholics (30 patients each)--identified by the MALT score--were examined. Clinical and laboratory investigations showed no connection between thiamine, riboflavin, or Vitamin B6 deficiency and development of the polyneuropathy. Neither the polyneuropathy nor the diminished sensory conduction velocity were related to malnutrition.
D, Claus +4 more
openaire +2 more sources
Two groups of alcoholics (30 patients each)--identified by the MALT score--were examined. Clinical and laboratory investigations showed no connection between thiamine, riboflavin, or Vitamin B6 deficiency and development of the polyneuropathy. Neither the polyneuropathy nor the diminished sensory conduction velocity were related to malnutrition.
D, Claus +4 more
openaire +2 more sources
Experientia, 1978
Rats which exhibited a preference for drinking a 6% w/v solution of ethanol in a free choice situation did not differ in their sensitivity to ethanol from animals exhibiting an aversion for ethanol, as measured by leaning rates in a T-maze task in which ethanol served as a discriminative stimulus.
openaire +2 more sources
Rats which exhibited a preference for drinking a 6% w/v solution of ethanol in a free choice situation did not differ in their sensitivity to ethanol from animals exhibiting an aversion for ethanol, as measured by leaning rates in a T-maze task in which ethanol served as a discriminative stimulus.
openaire +2 more sources
Acta Paediatrica, 1985
ABSTRACT. A 3‐year‐old boy with severe ethanol poisoning metabolised ethanol more rapidly than expected (0.33 g/l/hour or 7.16 mmol/l/hour) and recovered fully with conservative management. It is concluded that active elimination techniques are unnecessary for most children with acute ethanol poisoning, child
P J, Gibson, A J, Cant, T G, Mant
openaire +2 more sources
ABSTRACT. A 3‐year‐old boy with severe ethanol poisoning metabolised ethanol more rapidly than expected (0.33 g/l/hour or 7.16 mmol/l/hour) and recovered fully with conservative management. It is concluded that active elimination techniques are unnecessary for most children with acute ethanol poisoning, child
P J, Gibson, A J, Cant, T G, Mant
openaire +2 more sources
Acta Neurologica Scandinavica, 1983
Alterations in nervous system functioning following acute and chronic ethanol exposure have been studied in a great number of experimental investigations. Results from many of these investigations can be difficult to interpret, particularly since a variety of techniques and exposure models are employed.
openaire +2 more sources
Alterations in nervous system functioning following acute and chronic ethanol exposure have been studied in a great number of experimental investigations. Results from many of these investigations can be difficult to interpret, particularly since a variety of techniques and exposure models are employed.
openaire +2 more sources