Results 261 to 270 of about 478,578 (307)
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Catalytic oxidation of ethanol over tantalum oxide
Journal of Catalysis, 1972Abstract The reaction of ethanol and oxygen on tantalum oxide has been investigated at 250–320 °C. In the early stages of the reaction, the catalyst activity is rather low and ethanol undergoes dehydration as well as oxidation. However use of the catalyst for a long period causes a remarkable increase in the rate of acetaldehyde formation.
M. Legendre, D. Cornet
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Oxidative Esterification of Ethanol over Oxide Catalysts
Chemistry Letters, 1986Abstract Oxidative esterification of ethanol has been studied over various oxide catalysts. Acetaldehyde was easily formed by oxidative dehydrogenation, and reacted further with remained ethanol and oxygen to produce ester. Among single oxides, Bi2O3, MoO3, and Sb2O4 showed the highest selectivity and yield for ethyl acetate formation ...
Lianchi Wang +3 more
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Effects of ethanol on mitochondrial oxidations
Biochimica et Biophysica Acta (BBA) - General Subjects, 1964Abstract 1. 1. Effects of ethanol on mitochondrial oxidations in vitro have been studied. 2. 2. Ethanol in relatively low concentrations was found to stimulate the O 2 uptake of rat-liver mitochondria with β-hydroxybutyrate as the substrate. With pyruvate, α-ketoglutarate and succinate as substrate ethanol depressed the O 2 uptake. 3.
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Ethanol oxidation by intestinal microsomes: Increased activity after chronic ethanol administration
Life Sciences, 1979Abstract In the rat, chronic ethanol ingestion increases microsomal protein of the mucosal cells of the proximal small intestine, enhances cytochrome P-450 content and NADPH cytochrome-C-reductase activity. Intestinal oxidation of ethanol by a microsomal system is demonstrated as well as its enhancement after chronic ethanol administration.
H K, Seitz, M A, Korsten, C S, Lieber
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Ethanol oxidation in rat-liver mitochondria
Chemico-Biological Interactions, 1969Abstract (1) Ethanol is oxidized only slowly in rat-liver mitochondria. Addition of NAD, however, increases the oxidation rate to the level of the pyruvate oxidation. (2) Pyrazole, rotenone, antimycin A, and oligomycin are inhibitors of the mitochondrial ethanol oxidation. (3) The oxidation is dependent on the presence of ADP.
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Studies on Ethanol‐Brain Catalase Interaction: Evidence for Central Ethanol Oxidation
Alcoholism: Clinical and Experimental Research, 1991The purpose of the present investigation was to further study the relationship between ethanol and brain catalase in vivo. Rats were pretreated intraperitoneally (ip) with varying doses of ethanol or saline min prior to administration of cyanamide (0.68 mmol/kg; ip), 4‐hydroxypyrazole (1 mmol/kg; ip) or saline.
C M, Aragon, L M, Stotland, Z, Amit
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Industrial & Engineering Chemistry Research, 1993
With the Clean Air Act amendments of 1990 requiring increased use of oxygenated compounds such as alcohols and ethers in motor fuels, the problem of effectively controlling the emissions caused by burning these substances has become a more pressing issue.
Hariharan Rajesh, Umit S. Ozkan
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With the Clean Air Act amendments of 1990 requiring increased use of oxygenated compounds such as alcohols and ethers in motor fuels, the problem of effectively controlling the emissions caused by burning these substances has become a more pressing issue.
Hariharan Rajesh, Umit S. Ozkan
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Oxidative Stress and Ethanol Toxicity
2015Alcohol consumption coevolved with agrarian societies in the World. The benefits of alcohol are countered by the detriments it causes to society and to individual health. Indeed, one could argue that alcohol is the most common poison voluntarily consumed at toxic doses by the human population.
Juliane I. Beier, Gavin E. Arteel
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Oxidation of Ethanol on Sn-Mo Oxide Catalysts
Collection of Czechoslovak Chemical Communications, 1992To determine the acidic and basic properties of the title catalysts, the adsorption of NH3 and SO2 was compared using pulse method. It was found that this characteristics undergoes changes when the Sn-Mo catalyst is treated with aqueous potassium hydroxide solutions of different concentrations.
Magdy A. Wassel +2 more
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Ethanol‐Induced Oxidative Stress and Nutritional Status
Alcoholism: Clinical and Experimental Research, 1993The ability of dietary ethanol, administered over a 10‐day period, to elevate production rates of reactive oxygen species and to alter glutathione levels has been determined in both liver and cerebellum, a brain region known to be susceptible to ethanol‐induced damage. Two groups of ethanol‐consuming rats were used.
S C, Bondy, K R, Pearson
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