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Metabolism of ethidium bromide in rats.
Drug Metabolism and Disposition, 1981The biliary excretion products recovered after iv administration of ethidium bromide to rats were found to consist of: unchanged drug, 8-acetylethidium, and an 8-acetylated hydroxylated metabolite. Proofs of structure were obtained by field desorption mass spectroscopy and 1H NMR, which indicated that the latter metabolite is probably substituted at ...
C, Fraire, P, Lecointe, C, Paoletti
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Synthesis of 6-14C-ethidium bromide
Analytical Biochemistry, 1972Abstract The preparation of 6- 14 C-ethidium bromide of high specific radioactivity is described. The synthesis starts from 14 C-carboxyl-labeled benzoic acid and 4,4′-dinitro-2-aminodiphenyl. The crude compound was purified by chromatography on aluminum oxide.
F J, Loomeijer, A M, Kroon
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Ethidium bromide enhancement of frameshift mutagenesis caused by photoactivatable ethidium analogs
Mutation Research, 1979Ethidium azide analogs (3-amino-8-azido-ethidium monoazide and ethidium diazide) have been developed as photosensitive probes in order to analyze directly the reversible in vivo interactions of ethidium bromide. Our preliminary observations [11], relating the mutagenic potential of the monoazide analog of ethidium, have been extended and refined, using
L W, Yielding +3 more
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Binding of ethidium bromide to avian erythrocyte chromatin
Biochemical and Biophysical Research Communications, 1972Summary Ethidium bromide intercalation studies indicate that mature avian erythrocyte chromatin has 5.6 times fewer primary binding sites than deproteinized DNA. The number of both primary and secondary binding sites was increased upon removal of chromosomal proteins; among those, removal of avian erythrocyte specific histone V shows the highest ...
P F, Lurquin, V L, Seligy
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Ethidium bromide resistance and enhancement of mitochondrial recombination
Molecular and General Genetics MGG, 1974In some yeast cytoplasmic mutants, ethidium bromide resistance is expressed at the mitochondrial level. In homosexual crosses it leads to an enhanced recombination between two linked mitochondrial genes controlling chloramphenicol and erythromycin resistance or sensitivity, but recombination is still reciprocal.
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