Results 271 to 280 of about 142,409 (303)
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Neuroscience Letters, 1991
Application of 5-hydroxytryptamine (5-HT) to the lamprey spinal cord in vitro reversibly depressed the chemical component of excitatory post-synaptic potentials recorded intracellularly in motoneurons and evoked by stimulation of single reticulospinal Müller cells. The depression could be produced either by local application of small volumes of 10 mM 5-
Buchanan, James T., Grillner, Stan
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Application of 5-hydroxytryptamine (5-HT) to the lamprey spinal cord in vitro reversibly depressed the chemical component of excitatory post-synaptic potentials recorded intracellularly in motoneurons and evoked by stimulation of single reticulospinal Müller cells. The depression could be produced either by local application of small volumes of 10 mM 5-
Buchanan, James T., Grillner, Stan
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Brain Research, 1983
The spike peak and after-hyperpolarization of the action potential of bullfrog sympathetic ganglion cells were depressed during the late slow excitatory postsynaptic potential (EPSP). These changes in the action potential were mimicked by luteinizing hormone-releasing hormone (LH-RH), a neurotransmitter candidate for the late slow EPSP. LH-RH (5 microM)
T, Akasu, T, Nishimura, K, Koketsu
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The spike peak and after-hyperpolarization of the action potential of bullfrog sympathetic ganglion cells were depressed during the late slow excitatory postsynaptic potential (EPSP). These changes in the action potential were mimicked by luteinizing hormone-releasing hormone (LH-RH), a neurotransmitter candidate for the late slow EPSP. LH-RH (5 microM)
T, Akasu, T, Nishimura, K, Koketsu
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Neuroscience, 1998
In the CA1 area of the hippocampus, low frequency and tetanic conditioning stimuli are known to trigger long-term depression and potentiation of synaptic responses respectively and to produce irreversible excitatory postsynaptic potential/spike potentiation, i.e. an increase of the probability of discharge of the neurons.
C, Bernard, J, Pickering, H V, Wheal
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In the CA1 area of the hippocampus, low frequency and tetanic conditioning stimuli are known to trigger long-term depression and potentiation of synaptic responses respectively and to produce irreversible excitatory postsynaptic potential/spike potentiation, i.e. an increase of the probability of discharge of the neurons.
C, Bernard, J, Pickering, H V, Wheal
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Optoelectronic induction of field excitatory postsynaptic potential by organic photcapacitor
2021Optoelectronic induction of field excitatory postsynaptic potential by organic ...
Opančar, Aleksandar +3 more
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Development of excitatory and inhibitory postsynaptic potentials in the rat neocortex.
Perspectives on developmental neurobiology, 1995The postnatal development of synaptic potentials in the rat neocortex is characterized by the sequential appearance of functional excitatory and inhibitory synapses. Morphological and electrophysiological studies provided evidence that at early stages of development, pyramidal cells are extensively coupled to each other, presumably via gap junctions ...
B, Sutor, H J, Luhmann
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Excitatory Postsynaptic Potentials of Hippocampal Neurones and Their Low-Frequency Depression
1993The results obtained with recordings of aggregate (Chap. 1) and minimal FPs (Chap. 3) were considered as a support of the hypothesis suggesting that the basic LTP mechanism is a persistent increase in the efficacy of excitatory synapses. Nevertheless, it was necessary to test this hypothesis more directly with intracellular recordings, including ...
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Composite nature of the monosynaptic excitatory postsynaptic potential.
Journal of Neurophysiology, 1967openaire +2 more sources
Mechanisms shaping glutamate-mediated excitatory postsynaptic currents in the CNS
Current Opinion in Neurobiology, 1994Peter Jonas, Nelson Spruston
exaly