Results 141 to 150 of about 12,705,637 (166)

Acute Promyelocytic Leukemia: Pathophysiology, Diagnosis and Clinical Management. [PDF]

open access: yesHematol Rep
Abddaoui M   +3 more
europepmc   +1 more source

The Basic Principles of Pathophysiology of Venous Thrombosis. [PDF]

open access: yesInt J Mol Sci
Schulman S   +4 more
europepmc   +1 more source

Gene therapy as an innovative approach to the treatment of hemophilia B-a review. [PDF]

open access: yesJ Appl Genet
Wróblewska K   +5 more
europepmc   +1 more source

Warfarin-induced skin necrosis: a narrative review of clinical features, risk factors, and treatment strategies. [PDF]

open access: yesAnn Med Surg (Lond)
Mohamed Osman R   +4 more
europepmc   +1 more source

Laparoscopic Cholecystectomy for Acute Cholecystitis in a Patient with Acquired Hemophilia A: A Case Report of Perioperative Hemostatic Management with Plasma Exchange. [PDF]

open access: yesSurg Case Rep
Yasui K   +14 more
europepmc   +1 more source
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Phosphatidylserine Regulation of Coagulation Proteins Factor IXa and Factor VIIIa

The Journal of Membrane Biology, 2022
Blood coagulation is an intricate process, and it requires precise control of the activities of pro- and anticoagulant factors and sensitive signaling systems to monitor and respond to blood vessel insults. These requirements are fulfilled by phosphatidylserine, a relatively miniscule-sized lipid molecule amid the myriad of large coagulation proteins ...
Rinku Majumder
semanticscholar   +3 more sources

Nonasaccharide Inhibits Intrinsic Factor Xase Complex by Binding to Factor IXa and Disrupting Factor IXa–Factor VIIIa Interactions

Thrombosis and Haemostasis, 2019
A nonasaccharide (FG9) derived from natural fucosylated glycosaminoglycan (FG) is identified as a selective intrinsic factor Xase complex (FIXa-FVIIIa-Ca2+-phospholipid, FXase) inhibitor that possesses potential inhibition of venous thrombus in rats and ...
Chuang Xiao   +4 more
semanticscholar   +3 more sources

Factor IXa and Factor X Influence Factor VIIIa Stability and Inactivation Mechanisms In Vitro and In Vivo.

open access: yesBlood
Deficiency of (f)actor VIII causes hemophilia A and excess FVIII function increases venous thromboembolic risk. The phenotypic consequences of aberrant FVIII function underscore the importance of understanding mechanisms that downregulate (a)ctivated ...
Johnathan J Morris   +7 more
semanticscholar   +2 more sources

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