Results 111 to 120 of about 6,889,216 (287)

2015 Faculty Award Recipients

open access: yes, 2015
Faculty award recipients: Academy of Effective Teaching / Gina Brown, Brian Rawson -- Leadership in the Advancement of Teaching / Vinod Namboodiri -- Excellence in Teaching / Animesh Chakravarthy -- Excellence in Creative Activity / Aleksander Sternfeld ...
Faculty Senate
core   +1 more source

MITF maintains genome stability in nonmelanocyte lineages

open access: yesMolecular Oncology, EarlyView.
MITF is essential for melanocyte survival and acts as an oncogene in 10%–20% of melanomas. We show that MITF depletion causes genome instability in nonmelanocytic cells, leading to LATS2‐mediated P53 activation, cell cycle arrest, and apoptosis. This study highlights the role of MITF as a genome maintenance factor beyond the melanocyte lineage. Created
Drifa H. Gudmundsdottir   +13 more
wiley   +1 more source

2011 Faculty Award Recipients

open access: yes, 2011
Faculty award recipients: Academy of Effective Teaching / Jen-Chi Cheng -- Leadership in the Advancement of Teaching / Brien Bolin -- Excellence in Teaching / Kelly Anderson, Frank Rokosz -- Excellence in Creative Activity / Lynne Davis -- Excellence in ...
Faculty Senate
core   +1 more source

MODELING OF QUALITY IMPROVEMENT FACULTY MEMBERS IN EDUCATIONAL ORGANIZATIONS HIGHER EDUCATION

open access: yesМодернизация, инновация, развитие, 2015
The transition to a two-tier system of education, the introduction of the federal state educational standards of higher education, the requirements of the single European educational space are fundamentally changing the role of faculty in the ...
N. V. Sergeeva
doaj  

PAK1 activation drives divergent resistance mechanisms to aromatase inhibition and tamoxifen in a luminal: A breast cancer model

open access: yesMolecular Oncology, EarlyView.
Breast cancer remains a major cause of cancer death in women, frequently developing endocrine therapy resistance. This study demonstrates that upregulated p21‐activated kinase 1 (PAK1) activity drives resistance to tamoxifen and long‐term estrogen deprivation in ER+ breast cancer models.
Luisa Schwarzmüller   +10 more
wiley   +1 more source

CEACAM1 participation in breast cancer progression

open access: yesMolecular Oncology, EarlyView.
In invasive breast cancer (BC), CEACAM1 shifts from an apical to a uniform membranous/cytoplasmic pattern, or is lost, as tumors dedifferentiate, inversely tracking the Ki‐67 proliferative index. In MCF‐7 cells, only CEACAM1‐4L suppresses proliferation, repressing cell cycle and growth factor genes.
Mykola Lyndin   +3 more
wiley   +1 more source

2012 Faculty Award Recipients

open access: yes, 2012
Faculty award recipients: Academy of Effective Teaching / Elizabeth Behrman, Natalie Grant -- Leadership in the Advancement of Teaching / Carolyn Shaw -- Excellence in Teaching / Doris Burgert, Preethika Kumar -- Excellence in Creative Activity / Julie ...
Faculty Senate
core   +1 more source

Intrapatient tumour heterogeneity and clonal evolution in an autopsy study of metastatic salivary gland cancer

open access: yesMolecular Oncology, EarlyView.
Tumour heterogeneity and clonal evolution of metastatic salivary gland cancer were evaluated in two patients with adenoid carcinoma and one patient with myoepithelial carcinoma. Radiology‐guided autopsy enabled multi‐region sampling (total samples n = 149), followed by whole‐genome sequencing and phylogenetic reconstruction (17 tumour samples, 4–7 per ...
Gerben Lassche   +10 more
wiley   +1 more source

Faculty Senate Minutes, April 17, 2014 [PDF]

open access: yes, 2014
Valdosta State University Faculty Senate. "Faculty Senate Minutes, April 17, 2014." (Valdosta State University, 2014)
Faculty Senate, Valdosta State University
core  

Somatostatin receptor 4 (SSTR4) is a tumor suppressor in cutaneous and head & neck squamous cell carcinomas

open access: yesMolecular Oncology, EarlyView.
This study identifies somatostatin receptor 4 (Sstr4) as a critical tumor suppressor against skin and head/neck cancers (HNSCC, cSCC, and BCC). The loss of Sstr4 removes a check on cell growth, causing hyperactivation of the MAPK‐ERK signaling pathway (↑).
Ali Taqvi   +6 more
wiley   +1 more source

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