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Immunopathological effects of co-exposure to ochratoxin A and FAdV-4 in broilers
British Poultry Science1. Immunosuppressive disorders in poultry pose a significant threat to health and productivity. Ochratoxin A (OTA), a mycotoxin and fowl adenovirus serotype 4 (FAdV-4), the causative agent of hydropericardium syndrome and inclusion body hepatitis, are each known for their immunopathological effects.
M, Imran +6 more
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Protein profile of FAdV-4 based on SDS-PAGE and Western blot isolated from chickens in India
Comparative Immunology, Microbiology and Infectious Diseases, 2018Fowl adenovirus-4 (FAdV-4) causes hydropericardium syndrome (HPS) in poultry worldwide. An understanding of viral structural protein composition is important for developing novel immunodiagnostics and immunoprophylactics. Here we report isolation, culture, molecular and protein profile of FAdV-4 isolates recovered from HPS outbreaks in chicken in the ...
Prasenjit, Dhar +4 more
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Avian Diseases, 2021
Hepatitis hydropericardium syndrome (HHS) is an acute infectious disease caused by fowl adenovirus serotype-4 (FAdV-4), which mainly affects broilers aged 4-5 wk. During the winter of January 2021, a 32-day-old broiler flock (Cobb-500) suffered from unusually high mortality (15%) in the Alexandria Governorate, Egypt.
Hesham, Sultan +5 more
openaire +2 more sources
Hepatitis hydropericardium syndrome (HHS) is an acute infectious disease caused by fowl adenovirus serotype-4 (FAdV-4), which mainly affects broilers aged 4-5 wk. During the winter of January 2021, a 32-day-old broiler flock (Cobb-500) suffered from unusually high mortality (15%) in the Alexandria Governorate, Egypt.
Hesham, Sultan +5 more
openaire +2 more sources
Avian Diseases, 2010
By adapting a very virulent fowl adenovirus serotype 4 (FAdV-4) to a fibroblast cell line (QT35) instead of growing the virus in chicken embryo liver cells or chicken kidney cells, it was possible to attenuate the virus. Birds infected with the attenuated virus (FAdV-4/QT35) on the first day of life expressed no adverse clinical signs and no mortality.
Esther, Schonewille +3 more
openaire +2 more sources
By adapting a very virulent fowl adenovirus serotype 4 (FAdV-4) to a fibroblast cell line (QT35) instead of growing the virus in chicken embryo liver cells or chicken kidney cells, it was possible to attenuate the virus. Birds infected with the attenuated virus (FAdV-4/QT35) on the first day of life expressed no adverse clinical signs and no mortality.
Esther, Schonewille +3 more
openaire +2 more sources