Results 101 to 110 of about 24,724 (259)

Hevin Promotes Aging‐Related Cardiac Dysfunction via Facilitating Cardiac Inflammation in Male Mice

open access: yesAging Cell, Volume 25, Issue 2, February 2026.
Hevin released by iWAT into circulation stimulates cardiac macrophages through TLR4, inducing their polarization and CCL5 secretion, leading to worsened cardiac dysfunction and subsequent recruitment of additional inflammatory cells for pro‐inflammatory factor release.
Shi‐Yu Huang   +4 more
wiley   +1 more source

Dietary Protein Restriction Ameliorates Cardiac Inflammaging via AMPK‐ULK1‐Mediated Mitochondrial Quality Control

open access: yesAging Cell, Volume 25, Issue 2, February 2026.
Dietary protein restriction protects the aging heart in the context of obesity by limiting mitochondrial DNA leakage and suppressing cGAS–STING‐driven inflammation. Through AMPK–ULK1‐dependent mitophagy, protein restriction restores mitochondrial quality control, reduces cardiac remodeling, and preserves metabolic homeostasis during obesity‐associated ...
Wagner S. Dantas   +8 more
wiley   +1 more source

Extrasystoles on 24‐h Holter Monitoring During the First Year of Life

open access: yesActa Paediatrica, Volume 115, Issue 2, Page 370-377, February 2026.
ABSTRACT Aim To assess the natural course of extrasystoles and their association with tachyarrhythmias on 24‐h Holter monitoring during the first year of life. Methods A retrospective chart review was performed among newborns referred for 24‐h Holter monitoring at Tampere University Hospital from 2011 to 2017.
Asta Uusitalo   +5 more
wiley   +1 more source

P22.01: Fetal echocardiography and pregnancy outcome in Serbia [PDF]

open access: bronze, 2017
Svetlana Vrzić-Petronijević   +5 more
openalex   +1 more source

Heart matters: How glucose‐ and lipid‐modulating drugs remodel epicardial adipose tissue accumulation, inflammatory patterns and browning

open access: yesDiabetes, Obesity and Metabolism, Volume 28, Issue 2, Page 878-894, February 2026.
Abstract Epicardial adipose tissue (EAT) is a metabolically active visceral fat depot located between the myocardium and the visceral pericardium, exerting direct paracrine and vasocrine effects on the heart and coronary vessels. Under physiological conditions, EAT supports myocardial energy metabolism and thermoregulation through fatty acid supply and
Elisabeth Heuboeck   +2 more
wiley   +1 more source

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